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Triamcinolone preparations have been known to produce dermal atrophy. 3 The precise cause of the dermal atrophy produced by corticosteroid is unknown. Several mechanisms have been considered. The most accepted mechanism is the lymphatic spread of the corticosteroid suspension and resultant atrophy of the dermal and epidermal tissues. 1 Another proposed mechanism is the corticosteroidinduced vasoconstriction, especially when administered locally in high concentrations. This enhanced vasoconstriction may facilitate local thrombosis or embolization and capillary closure with resultant local tissue hypoxia. These effects then produce local tissue atrophy, or, in extreme cases, necrosis. 4

A review of the literature revealed many cases of atrophy following local injections of corticosteroid in the treatment of different lesions. 5, 6 A specific pattern of perilesional, streaklike linear atrophy or hypopigmentation, or both, after intralesional or intra-articular corticosteroid injection is rare. 1 Chodoroff et al, 7 however, reported a case of perilesional atrophy and hypopigmentation after intralesional corticosteroid injection in the treatment of de Quervain tendonitis.