作者
Suyash B Patil, Francois F Barbier, Jinfeng Zhao, Syed A Zafar, Muhammad Uzair, Yinglu Sun, Jingjing Fang, Maria‐Dolores Perez‐Garcia, Jessica Bertheloot, Soulaiman Sakr, Franziska Fichtner, Tinashe G Chabikwa, Shoujiang Yuan, Christine A Beveridge, Xueyong Li
发表日期
2022/4
期刊
New Phytologist
卷号
234
期号
1
页码范围
122-136
简介
- Shoot branching is regulated by multiple signals. Previous studies have indicated that sucrose may promote shoot branching through suppressing the inhibitory effect of the hormone strigolactone (SL). However, the molecular mechanisms underlying this effect are unknown.
- Here, we used molecular and genetic tools to identify the molecular targets underlying the antagonistic interaction between sucrose and SL.
- We showed that sucrose antagonizes the suppressive action of SL on tillering in rice and on the degradation of D53, a major target of SL signalling. Sucrose inhibits the gene expression of D3, the orthologue of the Arabidopsis F‐box MAX2 required for SL signalling. Overexpression of D3 antagonizes sucrose inhibition of D53 degradation and enables the SL inhibition of tillering under high sucrose. Sucrose prevents SL‐induced degradation of D14, the SL receptor involved in D53 degradation. In …
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