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1. Introduction acids and 2-monoacylglycerols for tissue utilization [3, 4]. Due to the large size of its substrates, the physiological site Atherosclerosis, the underlying cause of heart attacks, of LPL action is at the luminal surface of blood vessels, to stroke and peripheral vascular disease, is responsible for which the enzyme is attached via highly charged heparan over 50% of all deaths in developed countries. The disease sulphate proteoglycans (HSPG)[3, 4]. Mature LPL is can generally be viewed as a form of chronic inflammation secreted to the vascular endothelium from the parenchymal that is induced and perturbed by lipid accumulation and cells of the adipose and muscle tissues, its major sites of involves a number of components, including the damaged synthesis, and a variety of other tissues which have also endothelium, monocytes/macrophages, T cells, smooth been implicated as lesser, but significant …