关注
Aylin Camgöz, PhD
Aylin Camgöz, PhD
KITZ Heidelberg
在 kitz-heidelberg.de 的电子邮件经过验证
标题
引用次数
引用次数
年份
Mechanisms responsible for nilotinib resistance in human chronic myeloid leukemia cells and reversal of resistance
A Camgoz, EB Gencer, AU Ural, Y Baran
Leukemia & lymphoma 54 (6), 1279-1287, 2013
642013
RNAi profiling of primary human AML cells identifies ROCK1 as a therapeutic target and nominates fasudil as an antileukemic drug
M Wermke, A Camgoz, M Paszkowski-Rogacz, S Thieme, M von Bonin, ...
Blood, The Journal of the American Society of Hematology 125 (24), 3760-3768, 2015
632015
Inactivation of cancer mutations utilizing CRISPR/Cas9
C Gebler, T Lohoff, M Paszkowski-Rogacz, J Mircetic, D Chakraborty, ...
Journal of the National Cancer Institute 109 (1), djw183, 2017
472017
A single reporter mouse line for Vika, Flp, Dre, and Cre-recombination
M Karimova, O Baker, A Camgoz, R Naumann, F Buchholz, ...
Scientific reports 8 (1), 14453, 2018
342018
FOXR2 Stabilizes MYCN Protein and Identifies Non–MYCN-Amplified Neuroblastoma Patients With Unfavorable Outcome
F Schmitt-Hoffner, S van Rijn, UH Toprak, M Mauermann, F Rosemann, ...
Journal of Clinical Oncology 39 (29), 3217-3228, 2021
302021
Roles of ceramide synthase and ceramide clearence genes in nilotinib-induced cell death in chronic myeloidleukemia cells
A Camgoz, EB Gencer, AU Ural, F Avcu, Y Baran
Leukemia & lymphoma 52 (8), 1574-1584, 2011
272011
EZHIP: a new piece of the puzzle towards understanding pediatric posterior fossa ependymoma
A Jenseit, A Camgöz, SM Pfister, M Kool
Acta neuropathologica 143 (1), 1-13, 2022
262022
Macromolecular Changes in Nilotinib Resistant K562 Cells; an In vitro Study by Fourier Transform Infrared Spectroscopy
C Ceylan, A Camgoz, Y Baran
Technology in cancer research & treatment 11 (4), 333-344, 2012
192012
Comparative RNAi screens in isogenic human stem cells reveal SMARCA4 as a differential regulator
C Güneş, M Paszkowski-Rogacz, S Rahmig, S Khattak, A Camgöz, ...
Stem cell reports 12 (5), 1084-1098, 2019
112019
STK3 is a therapeutic target for a subset of acute myeloid leukemias
A Camgoz, M Paszkowski-Rogacz, S Satpathy, M Wermke, MV Hamann, ...
Oncotarget 9 (39), 25458, 2018
112018
CRISPR/Cas9 Screen in Gastric Cancer Patient‐Derived Organoids Reveals KDM1A‐NDRG1 Axis as a Targetable Vulnerability
J Mircetic, A Camgöz, M Abohawya, L Ding, J Dietzel, SG Tobar, ...
Small methods 7 (6), 2201605, 2023
92023
RNAi-Mediated Screen of Primary AML Cells Nominates MDM4 as a Therapeutic Target in NK-AML with DNMT3A Mutations
OA Sidorova, S Sayed, M Paszkowski-Rogacz, M Seifert, A Camgöz, ...
Cells 11 (5), 854, 2022
52022
3D genome mapping identifies subgroup-specific chromosome conformations and tumor-dependency genes in ependymoma
K Okonechnikov, A Camgöz, O Chapman, S Wani, DE Park, JM Hübner, ...
Nature Communications 14 (1), 2300, 2023
42023
A genome wide shRNA screen in primary human AML cells identifies ROCK1 as a novel therapeutic target
M Wermke, A Camgoz, M Paszkowski-Rogacz, S Thieme, M von Bonin, ...
Blood 122 (21), 170, 2013
22013
EPEN-07. USP7 IS AN INTERACTION PARTNER OF EZHIP AND POTENTIAL DRUGGABLE TARGET IN PFA EPENDYMOMAS
A Jenseit, A Camgöz, M Mauermann, N Mack, B Schwalm, J Mohr, ...
Neuro-Oncology 26 (Suppl 4), 0, 2024
2024
3D genome conformation analysis in ST-EPN-ZFTA ependymoma identifies RCOR2 as a potential target
A Camgoez, K Okonechnikov, O Chapman, M Maurmann, F Buchholz, ...
Cancer Research 82 (12_Supplement), 3886-3886, 2022
2022
EPEN-18. Oncogenic 3D genome conformations identify novel therapeutic targets in ependymoma
K Okonechnikov, A Camgöz, DE Park, O Chapman, JM Hübner, A Jenseit, ...
Neuro-Oncology 24 (Supplement_1), i42-i42, 2022
2022
Abstract A25: Evaluation of Drug Disposition in Supratentorial Ependymoma
J Benzel, M Sauter, N Mack, A Davis, J Weiss, P Uhl, J Burhenne, ...
Cancer Research 80 (14_Supplement), A25-A25, 2020
2020
Evaluation of Drug Disposition in Supratentorial Ependymoma
J Benzel, M Sauter, N Mack, A Davis, J Weiss, P Uhl, J Burhenne, ...
CANCER RESEARCH 80 (14), 54-55, 2020
2020
RNAi Screen in Primary AML Cells to Identify New Therapeutic Targets
A Camgöz
Technische Universität Dresden, 2018
2018
系统目前无法执行此操作,请稍后再试。
文章 1–20