| Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine M Dichgans, T Freilinger, G Eckstein, E Babini, B Lorenz-Depiereux, ... The Lancet 366 (9483), 371-377, 2005 | 1051 | 2005 |
| Rates of diffusional exchange between small cells and a measuring patch pipette M Pusch, E Neher Pflügers Archiv 411, 204-211, 1988 | 806 | 1988 |
| A chloride channel widely expressed in epithelial and non-epithelial cells A Thiemann, S Gründer, M Pusch, TJ Jentsch Nature 356 (6364), 57-60, 1992 | 672 | 1992 |
| Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II H Terlau, SH Heinemann, W Stühmer, M Pusch, F Conti, K Imoto, S Numa FEBS letters 293 (1-2), 93-96, 1991 | 589 | 1991 |
| Chloride/proton antiporter activity of mammalian CLC proteins ClC-4 and ClC-5 A Picollo, M Pusch Nature 436 (7049), 420-423, 2005 | 544 | 2005 |
| Regions involved in the opening of CIC-2 chloride channel by voltage and cell volume S Gründer, A Thiemann, M Pusch, TJ Jentsch Nature 360 (6406), 759-762, 1992 | 496 | 1992 |
| Gating of the voltage-dependent chloride channel CIC-0 by the permeant anion M Pusch, U Ludewig, A Rehfeldt, TJ Jentsch Nature 373 (6514), 527-531, 1995 | 398 | 1995 |
| CLC chloride channels and transporters: structure, function, physiology, and disease TJ Jentsch, M Pusch Physiological reviews 98 (3), 1493-1590, 2018 | 375 | 2018 |
| Two physically distinct pores in the dimeric CIC-0 chloride channel U Ludewig, M Pusch, TJ Jentsch Nature 383 (6598), 340-343, 1996 | 326 | 1996 |
| Molecular determinants of KCNQ (Kv7) K+ channel sensitivity to the anticonvulsant retigabine A Schenzer, T Friedrich, M Pusch, P Saftig, TJ Jentsch, J Grötzinger, ... Journal of Neuroscience 25 (20), 5051-5060, 2005 | 314 | 2005 |
| Heteromultimeric CLC chloride channels with novel properties C Lorenz, M Pusch, TJ Jentsch Proceedings of the National Academy of Sciences 93 (23), 13362-13366, 1996 | 286 | 1996 |
| Multimeric structure of ClC‐1 chloride channel revealed by mutations in dominant myotonia congenita (Thomsen). K Steinmeyer, C Lorenz, M Pusch, MC Koch, TJ Jentsch The EMBO journal 13 (4), 737-743, 1994 | 273 | 1994 |
| Myotonia caused by mutations in the muscle chloride channel gene CLCN1 M Pusch Human mutation 19 (4), 423-434, 2002 | 266 | 2002 |
| Properties of voltage‐gated chloride channels of the ClC gene family. TJ Jentsch, W Günther, M Pusch, B Schwappach The Journal of physiology 482 (suppl), 19-25, 1995 | 264 | 1995 |
| Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy M Schwake, M Pusch, T Kharkovets, TJ Jentsch Journal of Biological Chemistry 275 (18), 13343-13348, 2000 | 223 | 2000 |
| The muscle chloride channel ClC-1 has a double-barreled appearance that is differentially affected in dominant and recessive myotonia C Saviane, F Conti, M Pusch The Journal of general physiology 113 (3), 457-468, 1999 | 221 | 1999 |
| Mutations in dominant human myotonia congenita drastically alter the voltage dependence of the CIC-1 chloride channel M Pusch, K Steinmeyer, MC Koch, TJ Jentsch Neuron 15 (6), 1455-1463, 1995 | 219 | 1995 |
| Biophysical properties of acid-sensing ion channels (ASICs) S Gründer, M Pusch Neuropharmacology 94, 9-18, 2015 | 198 | 2015 |
| Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1 R Estévez, BC Schroeder, A Accardi, TJ Jentsch, M Pusch Neuron 38 (1), 47-59, 2003 | 196 | 2003 |
| Functional and structural conservation of CBS domains from CLC chloride channels R Estévez, M Pusch, C Ferrer‐Costa, M Orozco, TJ Jentsch The Journal of physiology 557 (2), 363-378, 2004 | 188 | 2004 |
Gianni ZifarelliUniversity of Oxford在 dpag.ox.ac.uk 的电子邮件经过验证
