| Cooperation of the IFT-A complex with the IFT-B complex is required for ciliary retrograde protein trafficking and GPCR import T Kobayashi, Y Ishida, T Hirano, Y Katoh, K Nakayama Molecular biology of the cell 32 (1), 45-56, 2021 | 48 | 2021 |
| Molecular basis of ciliary defects caused by compound heterozygous IFT144/WDR19 mutations found in cranioectodermal dysplasia Y Ishida, T Kobayashi, S Chiba, Y Katoh, K Nakayama Human Molecular Genetics 30 (3-4), 213-225, 2021 | 20 | 2021 |
| Molecular basis underlying the ciliary defects caused by IFT52 variations found in skeletal ciliopathies Y Ishida, K Tasaki, Y Katoh, K Nakayama Molecular Biology of the Cell 33 (9), ar83, 2022 | 6 | 2022 |
| Compound heterozygous IFT81 variations in a skeletal ciliopathy patient cause Bardet–Biedl syndrome-like ciliary defects K Tasaki, Z Zhou, Y Ishida, Y Katoh, K Nakayama Human Molecular Genetics 32 (19), 2887-2900, 2023 | 1 | 2023 |
Kazuhisa NakayamaKyoto University在 pharm.kyoto-u.ac.jp 的电子邮件经过验证
Kazuhisa NakayamaKyoto University在 pharm.kyoto-u.ac.jp 的电子邮件经过验证
Yohei KatohPtBio Inc./Hiroshima University Genome Editing Innovation Center在 hiroshima-u.ac.jp 的电子邮件经过验证