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Felix Wiedmann
Felix Wiedmann
University Hospital Heidelberg
在 med.uni-heidelberg.de 的电子邮件经过验证
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引用次数
引用次数
年份
Upregulation of K2P3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation
C Schmidt, F Wiedmann, N Voigt, XB Zhou, J Heijman, S Lang, V Albert, ...
Circulation 132 (2), 82-92, 2015
2052015
Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient …
C Schmidt, F Wiedmann, XB Zhou, J Heijman, N Voigt, A Ratte, S Lang, ...
European Heart Journal 38 (22), 1764-1774, 2017
962017
Altered atrial cytosolic calcium handling contributes to the development of postoperative atrial fibrillation
FE Fakuade, V Steckmeister, F Seibertz, J Gronwald, S Kestel, J Menzel, ...
Cardiovascular research 117 (7), 1790-1801, 2021
602021
Stretch-activated two-pore-domain (K2P) potassium channels in the heart: Focus on atrial fibrillation and heart failure
C Schmidt, F Wiedmann, SM Kallenberger, A Ratte, JS Schulte, B Scholz, ...
Progress in biophysics and molecular biology 130, 233-243, 2017
542017
Novel electrophysiological properties of dronedarone: inhibition of human cardiac two-pore-domain potassium (K2P) channels
C Schmidt, F Wiedmann, PA Schweizer, R Becker, HA Katus, D Thomas
Naunyn-Schmiedeberg's archives of pharmacology 385, 1003-1016, 2012
512012
Cloning, functional characterization, and remodeling of K2P3. 1 (TASK-1) potassium channels in a porcine model of atrial fibrillation and heart failure
C Schmidt, F Wiedmann, C Langer, F Tristram, P Anand, W Wenzel, ...
Heart Rhythm 11 (10), 1798-1805, 2014
492014
Inhibition of cardiac two-pore-domain K+ (K2P) channels–an emerging antiarrhythmic concept
C Schmidt, F Wiedmann, PA Schweizer, HA Katus, D Thomas
European Journal of Pharmacology 738, 250-255, 2014
472014
Therapeutic targeting of two-pore-domain potassium (K2P) channels in the cardiovascular system
F Wiedmann, C Schmidt, P Lugenbiel, I Staudacher, AK Rahm, C Seyler, ...
Clinical Science 130 (9), 643-650, 2016
462016
Cardiac expression and atrial fibrillation-associated remodeling of K2P2. 1 (TREK-1) K+ channels in a porcine model
C Schmidt, F Wiedmann, F Tristram, P Anand, W Wenzel, P Lugenbiel, ...
Life sciences 97 (2), 107-115, 2014
452014
Axial Tubule Junctions Activate Atrial Ca2+ Release Across Species
S Brandenburg, J Pawlowitz, FE Fakuade, D Kownatzki-Danger, T Kohl, ...
Frontiers in Physiology 9, 1227, 2018
442018
Extracellular vesicles mediate intercellular communication: Transfer of functionally active microRNAs by microvesicles into phagocytes
L Claßen, LO Tykocinski, F Wiedmann, C Birr, P Schiller, C Tucher, ...
European Journal of Immunology 47 (9), 1535-1549, 2017
412017
Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K2P) channels in murine disease models: focus on TASK-1
F Wiedmann, JS Schulte, B Gomes, MP Zafeiriou, A Ratte, F Rathjens, ...
Basic Research in Cardiology 113, 1-14, 2018
392018
Class I antiarrhythmic drugs inhibit human cardiac two-pore-domain K+ (K2P) channels
C Schmidt, F Wiedmann, PA Schweizer, R Becker, HA Katus, D Thomas
European journal of pharmacology 721 (1-3), 237-248, 2013
392013
Genetic Ablation of TASK-1 (Tandem of P Domains in a Weak Inward Rectifying K+ Channel–Related Acid-Sensitive K+ Channel-1) (K2P3.1) K+ Channels …
C Schmidt, F Wiedmann, C Beyersdorf, Z Zhao, I El-Battrawy, H Lan, ...
Circulation: Arrhythmia and Electrophysiology 12 (9), e007465, 2019
342019
Small Conductance Ca2 +-Activated K+ (SK) Channel mRNA Expression in Human Atrial and Ventricular Tissue: Comparison Between Donor, Atrial Fibrillation …
E Darkow, TT Nguyen, M Stolina, FA Kari, C Schmidt, F Wiedmann, ...
Frontiers in physiology 12, 650964, 2021
322021
Mechanosensitive TREK-1 two-pore-domain potassium (K2P) channels in the cardiovascular system
F Wiedmann, S Rinné, B Donner, N Decher, HA Katus, C Schmidt
Progress in biophysics and molecular biology 159, 126-135, 2021
302021
Functional characterization of zebrafish K2P18.1 (TRESK) two-pore-domain K+ channels
AK Rahm, F Wiedmann, J Gierten, C Schmidt, PA Schweizer, R Becker, ...
Naunyn-Schmiedeberg's archives of pharmacology 387, 291-300, 2014
302014
Anti-KCNQ1 K+ channel autoantibodies increase IKs current and are associated with QT interval shortening in dilated cardiomyopathy
J Li, C Seyler, F Wiedmann, C Schmidt, PA Schweizer, R Becker, ...
Cardiovascular research 98 (3), 496-503, 2013
292013
Treatment of atrial fibrillation with doxapram: TASK-1 potassium channel inhibition as a novel pharmacological strategy
F Wiedmann, C Beyersdorf, XB Zhou, M Kraft, A Paasche, N Jávorszky, ...
Cardiovascular Research 118 (7), 1728-1741, 2022
282022
Pharmacologic TWIK‐related acid‐sensitive K+ channel (TASK‐1) potassium channel inhibitor A293 facilitates acute cardioversion of paroxysmal atrial fibrillation in a porcine …
F Wiedmann, C Beyersdorf, X Zhou, A Büscher, M Kraft, J Nietfeld, ...
Journal of the American Heart Association 9 (10), e015751, 2020
282020
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