关注
Bruce Cronstein
Bruce Cronstein
NYU School of Medicine
在 nyumc.org 的电子邮件经过验证
标题
引用次数
引用次数
年份
Adenosine receptors: therapeutic aspects for inflammatory and immune diseases
G Haskó, J Linden, B Cronstein, P Pacher
Nature reviews Drug discovery 7 (9), 759-770, 2008
14142008
Adenosine 5′-triphosphate and adenosine as endogenous signaling molecules in immunity and inflammation
MJL Bours, ELR Swennen, F Di Virgilio, BN Cronstein, PC Dagnelie
Pharmacology & therapeutics 112 (2), 358-404, 2006
12962006
Adenosine: an endogenous regulator of innate immunity
G Haskó, BN Cronstein
Trends in immunology 25 (1), 33-39, 2004
10862004
A mechanism for the antiinflammatory effects of corticosteroids: the glucocorticoid receptor regulates leukocyte adhesion to endothelial cells and expression of endothelial …
BN Cronstein, SC Kimmel, RI Levin, F Martiniuk, G Weissmann
Proceedings of the National Academy of Sciences 89 (21), 9991-9995, 1992
9581992
Adenosine, an endogenous anti-inflammatory agent
BN Cronstein
Journal of applied Physiology 76 (1), 5-13, 1994
8611994
The antiinflammatory mechanism of methotrexate. Increased adenosine release at inflamed sites diminishes leukocyte accumulation in an in vivo model of inflammation.
BN Cronstein, D Naime, E Ostad
The Journal of clinical investigation 92 (6), 2675-2682, 1993
8471993
Low-dose methotrexate: a mainstay in the treatment of rheumatoid arthritis
BN Cronstein
Pharmacological reviews 57 (2), 163-172, 2005
7362005
Adenosine: a physiological modulator of superoxide anion generation by human neutrophils.
BN Cronstein, SB Kramer, G Weissmann, R Hirschhorn
The Journal of experimental medicine 158 (4), 1160-1177, 1983
5761983
Molecular action of methotrexate in inflammatory diseases
ESL Chan, BN Cronstein
Arthritis Research & Therapy 4, 1-8, 2002
5612002
Understanding the mechanisms of action of methotrexate
H Tian, BN Cronstein
Bull NYU Hosp Jt Dis 65 (3), 168-173, 2007
5482007
Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells.
BN Cronstein, RI Levin, J Belanoff, G Weissmann, R Hirschhorn
The Journal of clinical investigation 78 (3), 760-770, 1986
5461986
Methotrexate—how does it really work?
ESL Chan, BN Cronstein
Nature Reviews Rheumatology 6 (3), 175-178, 2010
4992010
The adenosine/neutrophil paradox resolved: human neutrophils possess both A1 and A2 receptors that promote chemotaxis and inhibit O2 generation, respectively.
BN Cronstein, L Daguma, D Nichols, AJ Hutchison, M Williams
The Journal of clinical investigation 85 (4), 1150-1157, 1990
4781990
Neutrophil adherence to endothelium is enhanced via adenosine A1 receptors and inhibited via adenosine A2 receptors.
BN Cronstein, RI Levin, M Philips, R Hirschhorn, SB Abramson, ...
Journal of immunology (Baltimore, Md.: 1950) 148 (7), 2201-2206, 1992
4761992
Methotrexate and its mechanisms of action in inflammatory arthritis
BN Cronstein, TM Aune
Nature Reviews Rheumatology 16 (3), 145-154, 2020
4442020
Adenosine; a physiologic modulator of superoxide anion generation by human neutrophils. Adenosine acts via an A2 receptor on human neutrophils.
BN Cronstein, ED Rosenstein, SB Kramer, G Weissmann, R Hirschhorn
Journal of immunology (Baltimore, Md.: 1950) 135 (2), 1366-1371, 1985
4441985
Colchicine alters the quantitative and qualitative display of selectins on endothelial cells and neutrophils.
BN Cronstein, Y Molad, J Reibman, E Balakhane, RI Levin, G Weissmann
The Journal of clinical investigation 96 (2), 994-1002, 1995
4421995
Methotrexate inhibits neutrophil function by stimulating adenosine release from connective tissue cells.
BN Cronstein, MA Eberle, HE Gruber, RI Levin
Proceedings of the National Academy of Sciences 88 (6), 2441-2445, 1991
4341991
Regulation of inflammation by adenosine
G Haskó, B Cronstein
Frontiers in immunology 4, 85, 2013
4162013
The adenosine-dependent angiogenic switch of macrophages to an M2-like phenotype is independent of interleukin-4 receptor alpha (IL-4Rα) signaling
CJ Ferrante, G Pinhal-Enfield, G Elson, BN Cronstein, G Hasko, S Outram, ...
Inflammation 36, 921-931, 2013
4072013
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