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Anis Hanna
Anis Hanna
Research Assistant Professor, Albert Einstein College of Medicine
在 einsteinmed.org 的电子邮件经过验证 - 首页
标题
引用次数
引用次数
年份
Inflammatory cytokines and chemokines as therapeutic targets in heart failure
A Hanna, NG Frangogiannis
Cardiovascular Drugs and Therapy 34 (6), 849-863, 2020
2322020
The role of the TGF-β superfamily in myocardial infarction
A Hanna, NG Frangogiannis
Frontiers in cardiovascular medicine 6, 140, 2019
2252019
Macrophage Smad3 protects the infarcted heart, stimulating phagocytosis and regulating inflammation
B Chen, S Huang, Y Su, YJ Wu, A Hanna, A Brickshawana, J Graff, ...
Circulation research 125 (1), 55-70, 2019
1112019
Protective effects of activated myofibroblasts in the pressure-overloaded myocardium are mediated through smad-dependent activation of a matrix-preserving program
I Russo, M Cavalera, S Huang, Y Su, A Hanna, B Chen, AV Shinde, ...
Circulation research 124 (8), 1214-1227, 2019
1052019
The role of Smad signaling cascades in cardiac fibrosis
A Hanna, C Humeres, NG Frangogiannis
Cellular signalling 77, 109826, 2021
682021
Properties and functions of fibroblasts and myofibroblasts in myocardial infarction
H Venugopal, A Hanna, C Humeres, NG Frangogiannis
Cells 11 (9), 1386, 2022
602022
Smad7 effects on TGF-β and ErbB2 restrain myofibroblast activation and protect from postinfarction heart failure
C Humeres, AV Shinde, A Hanna, L Alex, SC Hernández, R Li, B Chen, ...
The Journal of Clinical Investigation 132 (3), 2022
422022
The role of Smad2 and Smad3 in regulating homeostatic functions of fibroblasts in vitro and in adult mice
S Huang, B Chen, C Humeres, L Alex, A Hanna, NG Frangogiannis
Biochimica et Biophysica Acta (BBA)-Molecular Cell Research 1867 (7), 118703, 2020
352020
Validation of diagnostic criteria and histopathological characterization of cardiac rupture in the mouse model of nonreperfused myocardial infarction
A Hanna, AV Shinde, NG Frangogiannis
American Journal of Physiology-Heart and Circulatory Physiology 319 (5 …, 2020
262020
Collagen denaturation in the infarcted myocardium involves temporally distinct effects of MT1-MMP-dependent proteolysis and mechanical tension
A Hanna, AV Shinde, R Li, L Alex, C Humeres, P Balasubramanian, ...
Matrix Biology 99, 18-42, 2021
182021
Cardiac Pericytes Acquire a Fibrogenic Phenotype and Contribute to Vascular Maturation After Myocardial Infarction
L Alex, I Tuleta, SC Hernandez, A Hanna, H Venugopal, M Astorkia, ...
Circulation, 2023
72023
Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium
B Chen, R Li, SC Hernandez, A Hanna, K Su, AV Shinde, ...
Journal of Molecular and Cellular Cardiology 171, 1-15, 2022
72022
The role of the TGF-beta superfamily in myocardial infarction. Front Cardiovasc Med 6: 140
A Hanna, NG Frangogiannis
72019
The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction
J Li, R Li, I Tuleta, SC Hernandez, C Humeres, A Hanna, B Chen, ...
FASEB journal: official publication of the Federation of American Societies …, 2022
42022
The cell biological basis for primary unloading in acute myocardial infarction
A Hanna, NG Frangogiannis
International Journal of Cardiology 293, 45-47, 2019
32019
Diabetes Induces Cardiac Fibroblast Activation, Promoting a Matrix‐Preserving Nonmyofibroblast Phenotype, Without Stimulating Pericyte to Fibroblast Conversion
L Alex, I Tuleta, A Hanna, NG Frangogiannis
Journal of the American Heart Association 12 (6), e027463, 2023
22023
Protective effects of macrophage-specific integrin α5 in myocardial infarction are associated with accentuated angiogenesis
R Li, B Chen, A Kubota, A Hanna, C Humeres, SC Hernandez, Y Liu, ...
Nature communications 14 (1), 7555, 2023
12023
Abstract MP262: Endogenous Smad7 restrains myofibroblast activation and protects from post-infarction heart failure by suppressing tgf-beta signaling and by directly inhibiting …
C Humeres, AV Shinde, A Hanna, L Alex, S Conway, NG Frangogiannis
Circulation Research 129 (Suppl_1), AMP262-AMP262, 2021
12021
Smad7 Induction in Activated Infarct Myofibroblasts Protects From Adverse Remodeling by Restraining TGF-beta-Mediated and Receptor Tyrosine Kinase Signaling
CD Humeres, AV Shinde, A Hanna, S Conway, NG Frangogiannis
Circulation 142 (Suppl_3), A16793-A16793, 2020
12020
BAK contributes critically to necrosis and infarct generation during reperfused myocardial infarction
D Qin, XF Jia, A Hanna, J Lee, R Pekson, JW Elrod, JW Calvert, ...
Journal of Molecular and Cellular Cardiology 184, 1-12, 2023
2023
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