Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia–reperfusion in a rat model

Acta Physiologica, 2013 - Wiley Online Library
Aim Ischaemia–reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …
被引用次数：48 相关文章
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[HTML][HTML] Arginase inhibition improves coronary microvascular function and reduces infarct size following ischemia-reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung… - Acta physiologica …, 2013 - ncbi.nlm.nih.gov
Aim Ischemia-reperfusion injury is associated with reduced bioavailability of nitric oxide and
microvascular dysfunction. One emerging mechanism behind reduced nitric oxide …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia–reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung, D Berkowitz… - Acta Physiologica, 2013 - infona.pl
Aim Ischaemia–reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …

[引用][C] Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung, D Berkowitz… - Acta …, 2013 - cir.nii.ac.jp
Arginase inhibition improves coronary microvascular function and reduces infarct size following
ischaemia-reperfusion in a rat model | CiNii Research CiNii 国立情報学研究所 学術情報 …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model.

J Grönros, A Kiss, M Palmér, C Jung… - Acta …, 2013 - search.ebscohost.com
Aim Ischaemia-reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung… - Acta physiologica …, 2013 - pubmed.ncbi.nlm.nih.gov
Aim Ischaemia-reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischemia-reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung, D Berkowitz… - Acta …, 2013 - hero.epa.gov
AIM: Ischemia-reperfusion injury is associated with reduced bioavailability of nitric oxide and
microvascular dysfunction. One emerging mechanism behind reduced nitric oxide …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model.

J Grönros, A Kiss, M Palmér, C Jung… - Acta Physiologica …, 2013 - europepmc.org
Aim Ischaemia-reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model

J Grönros, A Kiss, M Palmér, C Jung… - Acta …, 2013 - pure.johnshopkins.edu
Aim: Ischaemia-reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …

Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model.

J Grönros, A Kiss, M Palmér, C Jung… - Acta Physiologica …, 2013 - europepmc.org
Aim Ischaemia-reperfusion injury is associated with reduced bioavailability of nitric oxide
(NO) and microvascular dysfunction. One emerging mechanism behind reduced NO …