Bolus intravenous injections of 100 micrograms/kg 17 beta-estradiol significantly decreased
the pressor responses to norepinephrine (NE; 0.3 microgram/kg) at the fourth, fifth, and sixth …

Estrogen (E2) can pharmacologically inhibit Ca2+ influx into vascular smooth muscle cells.
However, little is known about the ability of E2 to alter vascular Ca2+ channel expression …

Vascular actions of progesterone have been reported, independently of estrogen, affecting
both blood pressure and other aspects of the cardiovascular system. To study possible …

Objective: The acute effects of progesterone (P) on the cardiovascular system were studied
in vivo in anesthetized rats and in vitro. Design and Methods: 1) In vivo blood pressure …

Properties of [3H] nitrendipine binding, high K'and Ca"-Induced contractions and the
inhibition of high K+-induced contractions by verapamil were investigated in the uterine …

Abstract 1 The effect of in vivo oestrogen treatment on both 45Ca influx in myometrial strips
and the kinetics of [3H)-nitrendipine binding in the isolated plasma membrane (PM) was …

In order to elucidate the mechanisms by which estrogen treatment increases uterine
contractiliy, the effect of estrogen on Ca uptake was measured in the isolated rabbit uterus …

The acute effects of 17 beta-estradiol on bladder contractile function and on calcium currents
were evaluated in vitro on isolated guinea pig bladder strips and detrusor myocytes …

Progesterone induced rapid relaxation of KCl‐contracted tail artery helical strips from rats.
The effect was dose dependent, with an IC50 (inhibitory concentration which produces 50 …

OBJECTIVE: Our purpose was to study the mechanism by which 17β-estradiol modulates
contractile activity in isolated rings of omental artery from nonpregnant and pregnant …