Axonal growth inhibitors are released during traumatic injuries to the adult mammalian
central nervous system, including after spinal cord injury. These molecules accumulate at
the injury site and form a highly inhibitory environment for axonal regeneration. Among
these inhibitory molecules, myelin-associated inhibitors, including neurite outgrowth
inhibitor A, oligodendrocyte myelin glycoprotein, myelin-associated glycoprotein, chondroitin
sulfate proteoglycans and repulsive guidance molecule A are of particular importance. Due …