Poststroke recovery processes include restoration or compensation of function, respectively
functions initially lost or new functions acquired after an injury. Therapeutic interventions can
enhance these processes and/or reduce processes impeding regeneration. Numerous
experimental studies suggest great opportunities for such treatments, but the results from
recent large clinical trials using neuromodulators such as dopamine and fluoxetine are
disappointing. The reasons for this are manifold affecting forward translation of results from …