B cell deletion, anergy, and receptor editing in “knock in” mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain

Y Pewzner-Jung, D Friedmann, E Sonoda… - The Journal of …, 1998 - journals.aai.org
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B cell deletion, anergy, and receptor editing in" knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain

Y Pewzner-Jung, D Friedmann… - Journal of …, 1998 - weizmann.elsevierpure.com
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

[PDF][PDF] B Cell Deletion, Anergy, and Receptor Editing in “Knock In” Mice Targeted with a Germline-Encoded or Somatically Mutated Anti-DNA Heavy Chain

Y Pewzner-Jung, D Friedmann, E Sonoda, S Jung… - 1998 - Citeseer
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B cell deletion, anergy and receptor editing in" knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain.

Y Pewzner-Jung, D Friedmann, E Sonoda, S Jung… - 1998 - cabidigitallibrary.org
Mice in which germ-line or somatically mutated anti-DNA immunoglobulin heavy chains
were targeted to the heavy chain locus (" knock in" mice) were generated. The targeted …

B cell deletion, anergy, and receptor editing in" knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain

Y Pewzner-Jung, D Friedmann… - Journal of …, 1998 - … .esploro.exlibrisgroup.com
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B Cell Deletion, Anergy, and Receptor Editing in “Knock In” Mice Targeted with a Germline-Encoded or Somatically Mutated Anti-DNA Heavy Chain

Y Pewzner-Jung, D Friedmann, E Sonoda… - The Journal of …, 1998 - journals.aai.org
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B Cell Deletion, Anergy, and Receptor Editing in “Knock In” Mice Targeted with a Germline-Encoded or Somatically Mutated Anti-DNA Heavy Chain

Y Pewzner-Jung, D Friedmann, E Sonoda… - The Journal of …, 1998 - cir.nii.ac.jp
抄録< jats: title> Abstract</jats: title>< jats: p> To study the relative contributions of clonal
deletion, clonal anergy, and receptor editing to tolerance induction in autoreactive B cells …

[PDF][PDF] B Cell Deletion, Anergy, and Receptor Editing in “Knock In” Mice Targeted with a Germline-Encoded or Somatically Mutated Anti-DNA Heavy Chain

Y Pewzner-Jung, D Friedmann, E Sonoda, S Jung… - J …, 1998 - scholar.archive.org
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B cell deletion, anergy, and receptor editing in" knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain

Y Pewzner-Jung, D Friedmann… - Journal of …, 1998 - pubmed.ncbi.nlm.nih.gov
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …

B cell deletion, anergy, and receptor editing in" knock in" mice targeted with a germline-encoded or somatically mutated anti-DNA heavy chain

Y Pewzner-Jung, D Friedmann… - Journal of …, 1998 - weizmann.esploro.exlibrisgroup.com
To study the relative contributions of clonal deletion, clonal anergy, and receptor editing to
tolerance induction in autoreactive B cells and their dependence on B cell receptor affinity …