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SIRT3‐mediated CypD‐K166 deacetylation alleviates neuropathic pain by improving mitochondrial dysfunction and inhibiting oxidative stress

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu… - Oxidative Medicine …, 2022 - Wiley Online Library
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …
被引用次数：16 相关文章

SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress

B Yan, Q Liu, X Ding, Y Lin, X Jiao… - Oxidative medicine …, 2022 - pubmed.ncbi.nlm.nih.gov
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …
[PDF] scienceopen.com

[PDF][PDF] Research Article SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative …

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu, H Miao… - 2022 - scienceopen.com
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …

SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress.

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu… - … Medicine and Cellular …, 2022 - europepmc.org
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …

SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress

B Yan, Q Liu, X Ding, Y Lin, X Jiao… - Oxidative Medicine …, 2022 - search.proquest.com
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …
[HTML] nih.gov

[HTML][HTML] SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu… - … Medicine and Cellular …, 2022 - ncbi.nlm.nih.gov
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …

SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress.

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu… - … Medicine and Cellular …, 2022 - europepmc.org
Numerous studies have shown that mitochondrial dysfunction manifested by increased
mitochondrial permeability transition pore (mPTP) opening and reactive oxygen species …

[引用][C] SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative Stress

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu, H Miao… - Oxidative Medicine and … - Hindawi

[引用][C] Research Article SIRT3-Mediated CypD-K166 Deacetylation Alleviates Neuropathic Pain by Improving Mitochondrial Dysfunction and Inhibiting Oxidative …

B Yan, Q Liu, X Ding, Y Lin, X Jiao, Y Wu, H Miao… - 2022