[HTML][HTML] Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - Theranostics, 2019 - ncbi.nlm.nih.gov
Abstract Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
K Min-A, SH Kim, N Ryu, M Ji-Hyun, K Ye-Ri… - …, 2019 - search.proquest.com
Abstract Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - 2019 - ir.ymlib.yonsei.ac.kr
Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in endolymphatic sac,
cochlea and vestibule, are known to cause autosomal recessive sensorineural hearing loss …
cochlea and vestibule, are known to cause autosomal recessive sensorineural hearing loss …
[引用][C] Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - Theranostics, 2019 - cir.nii.ac.jp
Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct
functional roles of pendrin in normal hearing | CiNii Research CiNii 国立情報学研究所 学術 …
functional roles of pendrin in normal hearing | CiNii Research CiNii 国立情報学研究所 学術 …
[PDF][PDF] Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - scienceopen.com
Abstract Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
[PDF][PDF] Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - pdfs.semanticscholar.org
Abstract Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing.
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim, J Jung… - Theranostics, 2019 - europepmc.org
Abstract Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
endolymphatic sac, cochlea and vestibule, are known to cause autosomal recessive …
Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing
MA Kim, SH Kim, N Ryu, JH Ma, YR Kim… - …, 2019 - pubmed.ncbi.nlm.nih.gov
Rationale: Mutations of SLC26A4 that abrogate pendrin, expressed in endolymphatic sac,
cochlea and vestibule, are known to cause autosomal recessive sensorineural hearing loss …
cochlea and vestibule, are known to cause autosomal recessive sensorineural hearing loss …