… showed that postconditioning with inhaled carbon monoxide (… I/R damage through anti-apoptotic
and anti-inflammatory … and reperfusion and carbon monoxide postconditioning (I/R + CO…

… Carbon monoxide (CO) acts as a therapeutic agent against neural injury via its anti-apoptotic
effect. Here we hypothesized that low-dose CO inhalation can protect RGCs in a rat model …

… in apoptosis and neuroinflammation in … neuroinflammation ( 11 ). Emphasizing translational
validity, CO has previously been applied in humans with no signs of toxic effects by inhaling …

… biogenesis, programmed cell death (apoptosis), cellular proliferation, inflammation, and
innate immune responses. Inhaled CO is widely known as an inhalation hazard due to its rapid …

… been implicated in neuronal cell death and functional injuries … mediates a type of programmed
cell death termed pyroptosis (5). … Carbon monoxide (CO), which is a type of neurotoxic …

… inhalation of carbon monoxide itself, and third by the intravenous injection or oral intake of
any kind of carbon monoxide … of CO reduced tubular necrosis and apoptosis. On a molecular …

… anti-apoptosis pathways. In conclusion, we demonstrated that postconditioning with inhaled
high-dose H 2 can be a useful antioxidant, anti-inflammatory and anti-apoptosis agent that …

… Retinal ischemia induces apoptosis leading to … Pre- and postconditioning approaches with
the molybdenum-based, … antagonize neurodegeneration and neuroinflammation in the brain …

… of inhaled carbon monoxide [1] and carbon monoxide … inflammation and apoptosis leading
to reduced cell death [9, 10]. … after immediate ALF-186 postconditioning. Enucleation was …

… biliverdin, ferrous iron and carbon monoxide. Heme oxygenase-… ISO post-conditioning notably
decreases the infarct volume … Systemic and neuroinflammation can induce endothelial cell …