… overexpress PD-L1, to investigate the ability of B cells … that B cells showing high expression
of PD-L1 protect NOD mice against T1D and downregulate diabetogenic CD4 + T cells. …

… B cells to be the antigen-presenting cell (APC) subtype most efficiently supporting the expansion
of diabetogenic T cells (… The ability of NOD-PerIg B cells to respond to TLR stimulation …

… through their APC function activating ß-cell-reactive cytotoxic T-… The diabetogenic capacity
of B-lymphocytes in two BCR … in NOD-PerIg mice compared to NOD and 116C-NOD mice, …

… NOD mouse model of diabetes indicate that B cells are necessary for islet antigen presentation
to both CD4 and CD8 T cells … , which may have inhibited our ability to discern the two …

… of T cell receptor Vp genes preferentially utilized by the NOD … the ability of NOD APC to
activate immimoregulatory T cells … CDS* T cells essential for initiation of the diabetogenic process.…

… Preferential survival of memory B-lymphocytes and … capacity to present autoantigens to
diabetogenic T cells ..........… comes from its ability to serve as a subset of pathogenic APC…

… prevents the development of T cell mediated inflammation in … model to investigate the
immunomodulatory capacity of IL-33 … IL-33/IL-2 axis in CD11c + DC has the ability to preferentially …

… probably exert their effects by presenting islet autoantigens to diabetogenic T cells … In the
NOD mouse model, inhibition of the ability of B cells to present antigen by either MHC class I or …

… DCs, MΦs, B cells, mTECs, stromal cells) and may not all be found in a given type of APC.
(+… Although studies in NOD mice consistently point to a defective ability of splenic CD8α + DCs …

… functions suppressing diabetogenic T cells in NOD mice is … the APC capacity of B lymphocytes
remaining in NOD mice … MZ, B lymphocytes preferentially expanded in mice that received …