Voltage-gated Cav1. 2 and Cav1. 3 (L-type) Ca2+ channels regulate neuronal excitability,
synaptic plasticity, and learning and memory. Densin-180 (densin) is an excitatory synaptic …

Cav1 (L-type) channels and calmodulin-dependent protein kinase II (CaMKII) are key
regulators of Ca2+ signaling in neurons. CaMKII directly potentiates the activity of Cav1. 2 …

In neurons L-type calcium currents contribute to synaptic plasticity and to activity-dependent
gene regulation. The subcellular localization of CaV1. 2 and its association with upstream …

The primary function of dystrophin is to form a link between the cytoskeleton and the
extracellular matrix. In addition to this crucial structural function, dystrophin also plays an …

In neurons L-type calcium currents function in gene regulation and synaptic plasticity, while
excessive calcium influx leads to excitotoxicity and neurodegeneration. The major neuronal …

Cav1. 3 (L-type) voltage-gated Ca2+ channels have emerged as key players controlling
Ca2+ signals at excitatory synapses. Compared with the more widely expressed Cav1. 2 L …

This study describes the functional interaction between Cav3. 2 calcium channels and the
Epithelial Sodium Channel (ENaC). β-ENaC subunits showed overlapping expression with …

Ca V 1 L-type calcium channels are key to regulating neuronal excitability, with the range of
functional roles enhanced by interactions with calmodulin, accessory proteins, or CaMKII …

Facilitation and inactivation of P/Q-type Ca 2+ currents mediated by Ca 2+/calmodulin
binding to Ca V 2.1 channels contribute to facilitation and rapid depression of synaptic …

Presynaptic voltage-gated calcium channels provide calcium for synaptic vesicle exocytosis.
We show here that a green fluorescent protein–tagged α1 subunit of the Caenorhabditis …