Myocardial infarction (MI), the globally leading cause of heart failure, morbidity and mortality,
involves post-MI ventricular remodeling, a complex process including acute injury healing …

Acute myocardial infarction (MI) due to coronary artery occlusion is accompanied by a
pathological remodeling response that includes hypertrophic cardiac growth and fibrosis …

Acute myocardial infarction (AMI) induces unfavorable left ventricular remodeling (LVR), a
complex process that involves molecular, cellular, and geometric alterations leading to …

Myocardial ischaemia/reperfusion (I/R)-induced remodelling generally includes cell death
(necrosis and apoptosis), myocyte hypertrophy, angiogenesis, cardiac fibrosis, and …

Rationale Myocardial infarction (MI) triggers a dynamic microRNA response with the
potential of yielding therapeutic targets. Objective We aimed to identify novel aberrantly …

Background—MicroRNAs (miRs) participate in many cardiac pathophysiological processes,
including ischemia/reperfusion (I/R)-induced cardiac injury. Recently, we and others …

Myocardial infarction (MI) is one of the most frequent cardiac emergencies, with significant
potential for mortality. One of the major challenges of the post-MI healing response is that …

Cardiac fibrosis after myocardial infarction (MI) has been identified as a key factor in the
development of heart failure. Although dysregulation of microRNA (miRNA) is involved in …

MicroRNAs are key regulators of the cardiac response to injury. MiR‐100 has recently been
suggested to be involved in different forms of heart failure, but functional studies are lacking …

Ischemic heart disease is a form of congestive heart failure that is caused by insufficient
blood supply to the heart, resulting in a loss of viable tissue. In response to the injury, the …