Aneurysmal subarachnoid hemorrhage (aSAH) patients develop delayed cerebral ischemia
and delayed deficits (DCI) within 2 weeks of aneurysm rupture at a rate of approximately …

Lower mean hemoglobin (HGB) levels are associated with unfavorable outcome after
spontaneous subarachnoid hemorrhage (SAH). Currently, there is no cutoff level for mean …

Apoptosis is the term given to programmed cell death, which has been widely connected to
a number of intracranial pathologies including stroke, Alzheimer's disease, and more …

Background Haptoglobin (Hp) comprising 2 light (α) and 2 heavy (β) chains has an
antioxidant effect following free hemoglobin binding. Among 3 phenotypes—Hp1-1 (two α1) …

Cerebral vasospasm and ischemic damage are important causes of mortality and morbidity
in patients affected by aneurysmal subarachnoid hemorrhage (SAH). Recently, ip …

Object. From the moment an intracranial aneurysm ruptures, cerebral blood flow is impaired,
and this impairment mainly determines the outcome in patients who survive after the initial …

Ironside et al Perihematomal Edema and ICH 1627 common pathway of the coagulation
cascade. 39 Thrombin can also induce PHE formation independent of fibrinogen, which …

The first 72 h after aneurysmal subarachnoid hemorrhage (SAH) is a critical period for the
patient. Most of the deaths in the SAH patient population occur during this time, and a …

BACKGROUND Patients who survive aneurysmal subarachnoid hemorrhage (aSAH) are at
risk for delayed neurological deficits (DND) and cerebral infarction. In this exploratory cohort …

Objective To perform an individual patient–level data (IPLD) analysis and to determine the
relationship between haptoglobin (HP) genotype and outcomes after aneurysmal …