Autoimmune heparin‐induced thrombocytopenia (aHIT) indicates the presence in patients of
anti‐platelet factor 4 (PF4)–polyanion antibodies that are able to activate platelets strongly …

Introduction: Heparin-induced thrombocytopenia (HIT) is known for its strong association
with thrombosis and distinct pathogenesis involving anti-PF4/polyanion antibodies that …

INTRODUCTION Heparin-induced thrombocytopenia (HIT) is a distinct clinicopathologic
syndrome caused by platelet-activating antibodies that recognize complexes of platelet …

Heparin-induced thrombocytopenia (HIT) is an immune complication of heparin therapy
caused by antibodies to complexes of platelet factor 4 (PF4) and heparin. Pathogenic …

Heparin‐induced thrombocytopenia (HIT) is a clinical‐pathological disorder; thus, laboratory
testing for the pathogenic platelet‐activating antiplatelet factor 4 (PF 4)/heparin antibodies is …

Heparin-induced thrombocytopenia (HIT) is a potentially devastating immune mediated
adverse drug reaction caused by the emergence of antibodies that activate platelets in the …

Background Heparin-induced thrombocytopenia (HIT) is typically caused by platelet-
activating immunoglobulin G (IgG) antibodies (Abs) against platelet factor 4 (PF4) …

Thrombocytopenia following heparin administration can be associated with an immune
reaction, now referred to as heparin-induced thrombocytopenia. HIT is essentially a …

Heparin-induced thrombocytopenia (HIT) is a ÔclinicopathologicÕ syndrome (ie diagnosis
depends on both clinical and pathologic criteria being present)[1]. The ÔclinicalÕ criteria …

Heparin-induced thrombocytopenia (HIT) is caused by heparin-dependent antibodies that
usually recognize multimolecular complexes of platelet factor 4–heparin (PF4–H). HIT can …