Previous reports have demonstrated that excess zinc (Zn2+) released from nerve terminals
following cerebral ischemia causes brain injury. Therefore, the disturbance of Zn2+ …

Backgrounds Although zinc acts as a major regulator of neuronal physiology, its
dyshomeostasis may cause neuronal cell death. Hydrogen sulfide (H 2 S) has been …

Copper (Cu) is an essential trace element and acts as a redox cofactor for many enzymes;
however, excess Cu is toxic to cells. Hydrogen sulfide (H2S) is a well-known toxic gaseous …

Abstract Hydrogen sulfide (H 2 S), a well-known toxic gas, is regarded as endogenous
neuromodulator and plays multiple roles in the central nervous system under physiological …

Background Hydrogen sulfide (H2S) exhibits protective effects in various disease models
including cerebral ischemia–reperfusion (I/R) injury. Nonetheless, mechanisms and identity …

Cerebral hypoxia is one of the main causes of cerebral injury. This study was conducted to
investigate the potential protective effect of H2S in in vitro hypoxic models by subjecting SH …

Abstract Hydrogen sulfide (H 2 S), a well-known endogenous mediator, has been shown to
exert protective effects against neuronal damage caused by brain ischemia, but the …

Sodium azide (NaN3) is a chemical of rapidly growing commercial importance. It is very
acutely toxic and inhibits cytochrome oxidase (COX) by binding irreversibly to the heme …

ABSTRACT Hydrogen sulfide (H2S), which is a well‐known toxic gas, is found in relatively
high concentrations in the brain. Although a neuromodulatory role of H2S has been …

Alzheimer's disease (AD) is the most common neurodegenerative disorder, responsible for>
50% of all dementia cases. Sodium azide (NaN3) inhibits cytochrome oxidase by irreversibly …