Role of oxidative stress in ischemia–reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart
S Maddika, V Elimban, D Chapman… - Canadian journal of …, 2009 - cdnsciencepub.com
Ischemia–reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
[引用][C] Role of oxidative stress in ischemia―reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart
S MADDIKA, V ELIMBAN, D CHAPMAN… - Canadian journal of …, 2009 - pascal-francis.inist.fr
Role of oxidative stress in ischemia―reperfusion-induced alterations in myofibrillar ATPase
activities and gene expression in the heart CNRS Inist Pascal-Francis CNRS Pascal and …
activities and gene expression in the heart CNRS Inist Pascal-Francis CNRS Pascal and …
Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart.
S Maddika, V Elimban, D Chapman… - Canadian Journal of …, 2009 - europepmc.org
Ischemia-reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
Role of oxidative stress in ischemia--reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart
S Maddika, V Elimban, D Chapman… - Canadian Journal of …, 2009 - go.gale.com
Ischemia-reperfusion (IR) in the heart has been shown to produce myoffbriller remodeling
and depress [Ca. sup. 2+] sensitivity of myofilaments; however, the mechanisms for these …
and depress [Ca. sup. 2+] sensitivity of myofilaments; however, the mechanisms for these …
Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart
S Maddika, V Elimban… - Canadian journal of …, 2009 - pubmed.ncbi.nlm.nih.gov
Ischemia-reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
[PDF][PDF] Role of oxidative stress in ischemia–reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart1
S Maddika, V Elimban, D Chapman… - Can. J. Physiol …, 2009 - academia.edu
Ischemia–reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart.
S Maddika, V Elimban, D Chapman… - Canadian Journal of …, 2009 - search.ebscohost.com
Ischemia–reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart.
S Maddika, V Elimban, D Chapman… - Canadian Journal of …, 2009 - search.ebscohost.com
Ischemia–reperfusion (IR) in the heart has been shown to produce myofibrillar remodeling
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …
and depress Ca2+ sensitivity of myofilaments; however, the mechanisms for these …