Cell wall damage in Staphylococcus aureus induces a rapid genome-wide response,
referred to as the cell wall stress stimulon. This response is mediated by a two-component …

In Staphylococcus aureus the VraSR two-component system acts as a sentinel that can
rapidly sense cell wall peptidoglycan damage and coordinate a response to enhance the …

Background Staphylococcus aureus remains a medical challenge in the treatment of
bacterial infections. It has acquired resistance to commonly used antibiotics, and to those …

In Staphylococcus aureus, vancomycin‐resistance‐associated response regulator (VraR) is
a part of the VraSR two‐component system, which is responsible for activating a cell wall …

Abstract The Staphylococcus aureus Vancomycin-resistance-associated response regulator
VraR is known as an important response regulator, member of the VraTSR three-component …

Transcriptional profiling of Staphylococcus aureus treated with cell wall-active antibiotics
identified the 2-component system, VraSR, as one of the key players in response to …

Staphylococcus aureus remains a clinical scourge. Recent studies have revealed that S.
aureus is capable of mounting a response to antibiotics that target cell wall peptidoglycan …

The exposure of Staphylococcus aureus to a broad range of cell wall-damaging agents
triggers the induction of a cell wall stress stimulon (CWSS) controlled by the VraSR two …

Exposure of Staphylococcus aureus to cell wall inhibitors induces massive overexpression
of a number of genes, provided that the VraSR two-component sensory regulatory system is …

A two-component system consisting of the histidine kinase vancomycin-resistance-
associated sensor and the response regulator vancomycin-resistance-associated regulator …