Alzheimer's disease (AD), certainly the most widespread proteinopathy, has as classical
neuropathological hallmarks, two groups of protein aggregates: senile plaques and …

Alzheimer's disease (AD) is one of the most prevalent neurodegenerative diseases,
characterized by impaired cognitive function due to progressive loss of neurons in the brain …

Although amyloid beta (Aβ) peptide can promote tau pathology and its toxicity is
concurrently tau-dependent, the underlying mechanisms of the in vivo interplay of these …

Alzheimer's disease (AD) is a neurodegenerative disease with an increasing morbidity,
mortality, and economic cost. Plaques formed by amyloid beta peptide (Aβ) and …

It is widely recognized that Alzheimer's disease (AD) is a common type of progressive
neurodegenerative disorder that results in cognitive impairment over time. Approximately …

The pathology of Alzheimer's disease (AD) is characterized by amyloid plaques (aggregates
of amyloid-β (Aβ)) and neurofibrillary tangles (aggregates of tau) and is accompanied by …

Alzheimer's disease (AD) is the most frequent cause of age-related neurodegeneration and
cognitive impairment, and there are currently no broadly effective therapies. The underlying …

Mitochondrial dysfunction is involved in Alzheimer's disease (AD) pathogenesis.
Mitochondria have their own genetic material; however, most of their proteins (∼ 99%) are …

On the basis of not only the endosymbiotic theory of eukaryotic cell organization and
evolution but also of observations of transcellular communication via Tunneling NanoTubes …

Mitochondrial dysfunction has been widely implicated in Alzheimer´ s disease (AD) etiology.
Since mitochondria control a wide range of cellular processes the up-stream position of this …