The pituitary hormone, prolactin, triggers lactation in nursing mothers. Under nonlactating
conditions, prolactin secretion is suppressed by powerful inhibition from hypothalamic …

Milk production in the nursing mother is induced by the hormone prolactin. Its release from
the anterior pituitary is generally under tonic inhibition by neuroendocrine tuberoinfundibular …

Tuberoinfundibular dopamine (TIDA) neurons are the central regulators of prolactin (PRL)
secretion. Their extensive functional plasticity allows a change from low PRL secretion in the …

Neuroendocrine tuberoinfundibular dopamine (TIDA) neurons tonically inhibit pituitary
release of the hormone, prolactin. Through the powerful actions of prolactin in promoting …

Balance in the body9s hormonal axes depends on feedback onto neuroendocrine
hypothalamic neurons. This phenomenon involves transcriptional and biosynthetic effects …

The relationship between neuronal impulse activity and neurotransmitter release remains
elusive. This issue is especially poorly understood in the neuroendocrine system, with its …

Altered physiological states require neuronal adaptation. In late pregnancy and lactation, a
sub-population of the mouse hypothalamic tuberoinfundibular dopaminergic (TIDA) neurons …

How autoreceptors contribute to maintaining a stable output of rhythmically active neuronal
circuits is poorly understood. Here, we examine this issue in a dopamine population …

Lactation is a state of hyperprolactinemia resulting in part from suppressed
tuberoinfundibular dopaminergic (TIDA) neuronal activity. The suckling stimulus contributes …

Prolactin (PRL) secretion from the anterior pituitary is tonically inhibited by
tuberoinfundibular dopamine (TIDA) neurons in the arcuate nucleus of the hypothalamus …