β-Amyloid peptide 1–42 (Aβ1–42) is generated from amyloid precursor protein (APP) and
associated with neurodegeneration in Alzheimer's disease (AD). Aβ1–42 has been shown to …

Memory loss, synaptic dysfunction, and accumulation of amyloid β-peptides (Aβ) are major
hallmarks of Alzheimer's disease (AD). Downregulation of the nitric oxide/cGMP/cGMP …

Currently, no effective therapeutics exist for the treatment of incurable neurodegenerative
diseases such as Alzheimer's disease (AD). The cellular prion protein (PrPC) acts as a high …

Background Alzheimer's disease (AD) is characterized by a decline in cognitive function and
accumulation of amyloid-β peptide (Aβ) in extracellular plaques. Mutations in amyloid …

Alzheimer's disease (AD) is a senile dementia characterized by amyloid plaques,
neurofibrillary tangles, and synaptic and cell loss. The “amyloid cascade” hypothesis …

In Alzheimer disease (AD), the accumulation of amyloid beta (Aβ) begins decades before
cognitive symptoms and progresses from intraneuronal material to extracellular plaques. To …

Alzheimer's disease (AD) is a neurodegenerative disorder characterized by cognitive
symptoms of learning and memory deficits. Such cognitive impairments are attributed to …

Alzheimer's disease (AD) is the most common neurodegenerative disturbances. Dysfunction
of synaptic plasticity and decline in cognitive functions are the most prominent features of …

Chronic stress has been suggested to influence the pathogenesis of Alzheimer's disease
(AD); however, the mechanism underlying this influence remains unknown. In this study, we …

Objective: Intracellular amyloid β‐protein (Aβ) contributes to neurodegeneration in
Alzheimer disease (AD). Apomorphine (APO) is a dopamine receptor agonist for Parkinson …