Alzheimer's disease (AD) is a multifactorial pathology characterized by β-amyloid (Aβ)
deposits, Tau hyperphosphorylation, neuroinflammatory response, and cognitive deficit …

It was recently revealed that gut microbiota promote amyloid-beta (Aβ) burden in mouse
models of Alzheimer's disease (AD). However, the underlying mechanisms when using …

Severe amyloidosis and plaque-localized neuro-inflammation are key pathological features
of Alzheimer's disease (AD). In addition to astrocyte and microglial reactivity, emerging …

Alzheimer's disease (AD) is characterized by the accumulation of specific proteins in the
brain. A recent study revealed that manipulating gut microbiota (GM) significantly reduced …

Alzheimer's disease is the most common form of dementia in the western world, however
there is no cure available for this devastating neurodegenerative disorder. Despite clinical …

Emerging evidence suggests that the gut microbiome actively regulates cognitive functions
and that gut microbiome imbalance is associated with Alzheimer's disease (AD), the most …

Background Previous studies show that antibiotic-mediated (abx) alteration of the gut
microbiome (GMB) results in a reduction of amyloid beta (Aβ) plaques and proinflammatory …

The pathway leading from amyloid-β deposition to cognitive impairment is believed to be a
cornerstone of the pathogenesis of Alzheimer's disease (AD). However, what drives amyloid …

Gut microbiota is proven to be involved in the development of beta amyloid (Aβ) pathology in
Alzheimer's disease (AD). Since there are difficulties in translating microbiota findings based …

Evidence that the gut microbiota plays a key role in the pathogenesis of Alzheimer's disease
is already unravelling. The microbiota-gut-brain axis is a bidirectional communication …