Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén, A Johnsson… - Journal of cancer …, 2009 - Springer
Objective The aim of this study was to investigate the effect of epidermal growth factor
receptor (EGFR) blockade on cell survival and on downstream signalling pathways using …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2009 - search.proquest.com
The aim of this study was to investigate the effect of epidermal growth factor receptor (EGFR)
blockade on cell survival and on downstream signalling pathways using the monoclonal …

[引用][C] Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2008 - cir.nii.ac.jp
Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK
pathways correlate with the effect of cetuximab in head and neck and colon cancer cell lines …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2009 - search.ebscohost.com
The aim of this study was to investigate the effect of epidermal growth factor receptor (EGFR)
blockade on cell survival and on downstream signalling pathways using the monoclonal …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2008 - europepmc.org
Objective The aim of this study was to investigate the effect of epidermal growth factor
receptor (EGFR) blockade on cell survival and on downstream signalling pathways using …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2009 - portal.research.lu.se
OBJECTIVE: The aim of this study was to investigate the effect of epidermal growth factor
receptor (EGFR) blockade on cell survival and on downstream signalling pathways using …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén… - Journal of Cancer …, 2009 - lup.lub.lu.se
OBJECTIVE: The aim of this study was to investigate the effect of epidermal growth factor
receptor (EGFR) blockade on cell survival and on downstream signalling pathways using …

[引用][C] Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and …

T YAMATODANI, L EKBLAD… - Journal of cancer …, 2009 - pascal-francis.inist.fr
Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK
pathways correlate with the effect of cetuximab in head and neck and colon cancer cell lines …

Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and colon …

T Yamatodani, L Ekblad, E Kjellén, A Johnsson… - Journal of Cancer …, 2009 - infona.pl
Objective The aim of this study was to investigate the effect of epidermal growth factor
receptor (EGFR) blockade on cell survival and on downstream signalling pathways using …

[引用][C] Epidermal growth factor receptor status and persistent activation of Akt and p44/42 MAPK pathways correlate with the effect of cetuximab in head and neck and …

T Yamatodani, L Ekblad, E Kjellén, A Johnsson… - Journal of Cancer …, 2009 - Springer