S100A4/non-muscle myosin II signaling regulates epithelial-mesenchymal transition and stemness in uterine carcinosarcoma
M Tochimoto, Y Oguri, M Hashimura, R Konno… - Laboratory …, 2020 - nature.com
Uterine carcinosarcoma (UCS) represents a true example of cancer associated with
epithelial-mesenchymal transition (EMT), which exhibits cancer stem cell (CSC)-like traits …
epithelial-mesenchymal transition (EMT), which exhibits cancer stem cell (CSC)-like traits …
[HTML][HTML] S100A4/nonmuscle myosin IIA/p53 axis contributes to aggressive features in ovarian high-grade serous carcinoma
A Hiruta, Y Oguri, A Yokoi, T Matsumoto, Y Oda… - The American journal of …, 2020 - Elsevier
S100A4 is a small calcium-binding protein that exerts its biological functions by interacting
with nonmuscle myosin IIA (NMIIA) and p53. Although S100A4 promotes metastasis in …
with nonmuscle myosin IIA (NMIIA) and p53. Although S100A4 promotes metastasis in …
[HTML][HTML] S100A4 promotes endometrial cancer progress through epithelial-mesenchymal transition regulation
T Hua, S Liu, X Xin, L Cai, R Shi, S Chi… - Oncology …, 2016 - spandidos-publications.com
Epithelial-mesenchymal transition (EMT) is a major cause of endometrial cancer (EC) to
initiate invasion and metastasis. S100A4, a calcium-binding protein, is implicated in …
initiate invasion and metastasis. S100A4, a calcium-binding protein, is implicated in …
S100A4 accelerates the proliferation and invasion of endometrioid carcinoma and is associated with the “MELF” pattern
S Tahara, S Nojima, K Ohshima, Y Hori… - Cancer …, 2016 - Wiley Online Library
Endometrioid carcinoma (EC) is one of the most common malignancies of the female genital
system. Although the behavior of EC ranges from an excellent prognosis to aggressive …
system. Although the behavior of EC ranges from an excellent prognosis to aggressive …
Upregulation of S100A6 in patients with endometriosis and its role in ectopic endometrial stromal cells
Y Peng, J Lin, J Ma, K Lin, K Xu, J Lin - Gynecological …, 2018 - Taylor & Francis
S100 calcium-binding protein A6 (S100A6) is up-regulated in many malignancies and
overexpression of S100A6 has been identified associated with proliferation, migration and …
overexpression of S100A6 has been identified associated with proliferation, migration and …
S100B mediates stemness of ovarian cancer stem-like cells through inhibiting p53
T Yang, J Cheng, Y Yang, W Qi, Y Zhao, H Long… - Stem …, 2017 - academic.oup.com
S100B is one of the members of the S100 protein family and is involved in the progression of
a variety of cancers. Ovarian cancer is driven by cancer stem-like cells (CSLCs) that are …
a variety of cancers. Ovarian cancer is driven by cancer stem-like cells (CSLCs) that are …
[HTML][HTML] Identification and characterization of a subpopulation of CD133+ cancer stem‐like cells derived from SK-UT-1 cells
J Gao, T Yang, X Wang, Y Zhang, J Wang… - Cancer Cell …, 2021 - Springer
Background Uterine leiomyosarcoma (ULMS) is a malignant tumor found in the smooth
muscle lining the walls of the uterus. Cancer stem cells (CSCs) are responsible for …
muscle lining the walls of the uterus. Cancer stem cells (CSCs) are responsible for …
S100P is overexpressed in squamous cell and Adenosquamous carcinoma subtypes of endometrial Cancer and promotes Cancer cell proliferation and invasion
ABSTRACT S100P is known to affect tumor development and metastasis of various cancers,
but its role in endometrial cancer is unclear. We reported that S100P expression was …
but its role in endometrial cancer is unclear. We reported that S100P expression was …
[HTML][HTML] Functional interaction between S100A1 and MDM2 may modulate p53 signaling in normal and malignant endometrial cells
M Nakagawa, S Higuchi, M Hashimura, Y Oguri… - BMC cancer, 2022 - Springer
Background S100A1 expression is deregulated in a variety of human malignancies, but its
role in normal and malignant endometrial cells is unclear. Methods We used endometrial …
role in normal and malignant endometrial cells is unclear. Methods We used endometrial …
[HTML][HTML] S100A6 promotes the proliferation and migration of cervical cancer cells via the PI3K/Akt signaling pathway
A Li, Y Gu, X Li, H Sun, H Zha, J Xie… - Oncology …, 2018 - spandidos-publications.com
Cervical cancer is the second most common gynecological cancer worldwide and remains
one of the leading causes of cancer-associated mortality among women. S100A6 has been …
one of the leading causes of cancer-associated mortality among women. S100A6 has been …