Nitric oxide (NO) is reported to cause neuronal damage through various mechanisms. The
present study tests the hypothesis that NO synthase inhibition by Nω‐nitro‐l‐arginine …

Hypoxia-induced brain cell membrane lipid peroxidation can be caused by free radicals that
are produced during hypoxia. Recently, the production of nitric oxide (NO), a free radical …

Previous studies have shown that brain tissue hypoxia results in increased N-methyl-D-
aspartate (NMDA) receptor activation and receptor-mediated increase in intracellular …

Previously, we have shown that hypoxia results in increased generation of nitric oxide free
radicals in the cerebral cortex of newborn piglets that may be due to up-regulation of nitric …

Nitric oxide (NO) produced by inducible NO synthase contributes to ischemic brain damage.
However, the role of inducible NO synthase-derived NO on neonatal hypoxic-ischemic …

To determine the role of nitric oxide production during hypoxia, the presence of two forms of
neuronal nitric oxide synthase, cytosolic (cNOS) and membrane-bound (memNOS), in …

We measured the changes in nitric oxide (NO) metabolites in the brains of neonatal rats with
hypoxic–ischemic damage. There were two peaks of NO metabolites in the lesioned side of …

We evaluated the neuroprotective effect of the nitric oxide synthesis inhibitor, N G-nitro-L-
arginine in a neonatal hypoxic-ischemic rat model. Unilateral hypoxic-ischemic injury was …

The activity of nitric oxide synthase (NOS) was measured in homogenates from cortex,
striatum, hippocampus, cerebellum, pons, thalamus and midbrain of the brain of newborn …

The present study was designed to examine the effects of inhibition of nitric oxide synthase
on cerebral energy metabolism after hypoxia-ischemia in newborn piglets. Ten 1-to 3-d-old …