The role of the neural messenger nitric oxide (NO) in cerebral ischaemia has been
investigated extensively in the past decade. NO may play either a protective or destructive …

Nitrogen monoxide (NO) has recently emerged as an important mediator of cellular and
molecular events which impacts the pathophysiology of cerebral ischemia. Although …

There is increasing evidence that nitric oxide (NO), a free radical that can act both as a
signaling molecule and a neurotoxin, is involved in the mechanisms of cerebral ischemia …

Nitric oxide (NO) is a new intercellular messenger that occurs naturally in the brain without
causing overt toxicity. Yet, NO has been implicated as a mediator of cell death in cell death …

Nitric oxide (NO) has several essential roles in mammals, but unregulated NO production
can cause cell death through oxidative stress, disrupted energy metabolism, DNA damage …

A large body of evidence has appeared over the last 6 years suggesting that nitric oxide
biosynthesis is a key factor in the pathophysiological response of the brain to hypoxia …

Ischaemic stroke is the second or third leading cause of death in developed countries. In the
last two decades substantial research and efforts have been made to understand the …

Background and Purpose—Excessive release of nitric oxide (NO) has been implicated in the
pathophysiology of neurodegeneration in ischemic stroke. We compared intracerebral …

Nitric oxide (NO) is a key signalling molecule in the regulation of cerebral blood flow. This
review summarises current evidence regarding the role of NO in the regulation of cerebral …

The original observation that inhibitors of nitric oxide (NO) synthesis can antagonize
glutamate toxicity in cell culture has led to extensive investigation of the role of NO in the …