The roles of MCPyV early region in IL-17A-mediated inflammation and autophagy
J Miao - 2021 - theses.hal.science
Merkel cell carcinoma (MCC) is a rare aggressive skin malignancy that affects
predominantly elderly and immunocompromised patients. Merkel cell polyomavirus …
predominantly elderly and immunocompromised patients. Merkel cell polyomavirus …
The MCPyV st-ag and IL33/ST2 axis: critical role in merkel cell carcinoma
K Rasheed, S Fismen, Ø Grimstad, B Sveinbjørnsson… - Cancer Research, 2019 - AACR
Abstract Merkel Cell polyomavirus (MCPyV) is a causal factor in Merkel Cell Carcinoma
(MCC). The oncogenic potential is mediated through its viral oncoproteins large T-antigen …
(MCC). The oncogenic potential is mediated through its viral oncoproteins large T-antigen …
Connecting Viral Oncoproteins to microRNA, Autophagy and Metabolism in Merkel Cell Carcinoma
S Kumar - 2019 - search.proquest.com
Merkel cell carcinoma (MCC) is an aggressive type of skin cancer. Around 80% of MCCs
harbor an integrated Merkel cell polyomavirus (MCPyV) genome with a mutation in the large …
harbor an integrated Merkel cell polyomavirus (MCPyV) genome with a mutation in the large …
Merkel cell polyomavirus oncoproteins induce microRNAs that suppress multiple autophagy genes
Viruses can inhibit host autophagy through multiple mechanisms, and evasion of autophagy
plays an important role in immune suppression and viral oncogenesis. Merkel cell …
plays an important role in immune suppression and viral oncogenesis. Merkel cell …
[HTML][HTML] Merkel cell polyomavirus small T antigen promotes pro-glycolytic metabolic perturbations required for transformation
Merkel cell polyomavirus (MCPyV) is an etiological agent of Merkel cell carcinoma (MCC), a
highly aggressive skin cancer. The MCPyV small tumor antigen (ST) is required for …
highly aggressive skin cancer. The MCPyV small tumor antigen (ST) is required for …
[HTML][HTML] The Merkel Cell Polyomavirus T-Antigens and IL-33/ST2-IL1RAcP Axis: Possible Role in Merkel Cell Carcinoma
K Rasheed, U Moens, B Policastro, JI Johnsen… - International Journal of …, 2022 - mdpi.com
Merkel cell polyomavirus (MCPyV) is a causal factor in Merkel cell carcinoma (MCC). The
oncogenic potential is mediated through its viral oncoproteins large T-antigen (LT) and small …
oncogenic potential is mediated through its viral oncoproteins large T-antigen (LT) and small …
[HTML][HTML] Characterization of molecular mechanisms driving Merkel cell polyomavirus oncogene transcription and tumorigenic potential
JF Yang, W Liu, J You - PLoS Pathogens, 2023 - journals.plos.org
Merkel cell polyomavirus (MCPyV) is associated with approximately 80% of cases of Merkel
cell carcinoma (MCC), an aggressive type of skin cancer. The incidence of MCC has tripled …
cell carcinoma (MCC), an aggressive type of skin cancer. The incidence of MCC has tripled …
Merkel cell polyomavirus small tumour antigen activates the p38 MAPK pathway to enhance cellular motility
Merkel cell carcinoma (MCC) is an aggressive skin cancer with high rates of recurrence and
metastasis. Merkel cell polyomavirus (MCPyV) is associated with the majority of MCC cases …
metastasis. Merkel cell polyomavirus (MCPyV) is associated with the majority of MCC cases …
[HTML][HTML] CCL17/TARC and CCR4 expression in Merkel cell carcinoma
K Rasheed, I Abdulsalam, S Fismen, Ø Grimstad… - Oncotarget, 2018 - ncbi.nlm.nih.gov
Merkel cell carcinoma (MCC) is a rare, highly aggressive neuroendocrine skin cancer. In
more than 80% of the cases, Merkel cell polyomavirus (MCPyV) is a causal factor. The …
more than 80% of the cases, Merkel cell polyomavirus (MCPyV) is a causal factor. The …
Understanding the role of Merkel cell polyomavirus oncoproteins in the cellular transformation of mammalian Merkel cells
LM Knight - 2014 - etheses.whiterose.ac.uk
Merkel cell carcinoma (MCC) is a highly aggressive skin cancer of neuroendocrine origin
with a high propensity for metastasis through the dermal lymphatic system. In 2008, Merkel …
with a high propensity for metastasis through the dermal lymphatic system. In 2008, Merkel …