[HTML][HTML] Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1) …
RS Nagalingam, NR Sundaresan, M Noor… - Journal of Biological …, 2014 - ASBMB
Understanding the regulation of cardiac fibrosis is critical for controlling adverse cardiac
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent …
RS Nagalingam, NR Sundaresan… - The Journal of …, 2014 - pubmed.ncbi.nlm.nih.gov
Understanding the regulation of cardiac fibrosis is critical for controlling adverse cardiac
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent …
RS Nagalingam, NR Sundaresan, M Noor… - The Journal of …, 2014 - europepmc.org
Understanding the regulation of cardiac fibrosis is critical for controlling adverse cardiac
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
[PDF][PDF] Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor (TGF 1) …
RS Nagalingam, NR Sundaresan, M Noor… - THE JOURNAL OF …, 2014 - researchgate.net
Background: MicroRNA-378, a cardiomyocyte-specific miRNA, is down-regulated during
heart failure. Results: miR-378 inhibition induced TGFß1 expression, which correlated with …
heart failure. Results: miR-378 inhibition induced TGFß1 expression, which correlated with …
[HTML][HTML] Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor β (TGFβ1) …
RS Nagalingam, NR Sundaresan, M Noor… - Journal of Biological …, 2014 - Elsevier
Understanding the regulation of cardiac fibrosis is critical for controlling adverse cardiac
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
[HTML][HTML] Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor β (TGFβ1) …
RS Nagalingam, NR Sundaresan, M Noor… - The Journal of …, 2014 - ncbi.nlm.nih.gov
Results: miR-378 inhibition induced TGFβ1 expression, which correlated with the activity of c-
Fos, c-Jun, and Ras. Conditioned media of miR-378-depleted myocytes induced fibroblast …
Fos, c-Jun, and Ras. Conditioned media of miR-378-depleted myocytes induced fibroblast …
Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent …
RS Nagalingam, NR Sundaresan, M Noor… - The Journal of …, 2014 - europepmc.org
Understanding the regulation of cardiac fibrosis is critical for controlling adverse cardiac
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …
remodeling during heart failure. Previously we identified miR-378 as a cardiomyocyte …