[HTML][HTML] Epithelial-mesenchymal plasticity induced by discontinuous exposure to TGFβ1 promotes tumour growth
Simple Summary In this manuscript, we used a non-genetically manipulated EMT/MET cell
line model to demonstrate that epithelial mesenchymal plasticity occurring in normal cells …
line model to demonstrate that epithelial mesenchymal plasticity occurring in normal cells …
[PDF][PDF] Epithelial-mesenchymal plasticity induced by
D Huntsman, C Oliveira - scholar.archive.org
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to 27
cancer progression. We hypothesize that EMT followed by MET promotes cell population 28 …
cancer progression. We hypothesize that EMT followed by MET promotes cell population 28 …
Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth.
M Santos, M Ferreira, P Oliveira… - Biology (2079 …, 2022 - search.ebscohost.com
Abstract Simple Summary: In this manuscript, we used a non-genetically manipulated
EMT/MET cell line model to demonstrate that epithelial mesenchymal plasticity occurring in …
EMT/MET cell line model to demonstrate that epithelial mesenchymal plasticity occurring in …
[HTML][HTML] Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - mdpi.com
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - …, 2022 - search.proquest.com
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
[HTML][HTML] Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - Biology, 2022 - ncbi.nlm.nih.gov
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
Epithelial-mesenchymal plasticity induced by discontinuous exposure to TGFβ1 promotes tumour growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - bioRxiv, 2021 - biorxiv.org
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - 2022 - agris.fao.org
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth.
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - Biology, 2022 - europepmc.org
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
Epithelial-Mesenchymal Plasticity Induced by Discontinuous Exposure to TGFβ1 Promotes Tumour Growth
M Santos, M Ferreira, P Oliveira, N Mendes, A André… - 2022 - open.library.ubc.ca
Transitions between epithelial and mesenchymal cellular states (EMT/MET) contribute to
cancer progression. We hypothesize that EMT followed by MET promotes cell population …
cancer progression. We hypothesize that EMT followed by MET promotes cell population …