Differential neuroprotection by cyclosporin A and FK506 following ischemia corresponds with differing abilities to inhibit calcineurin and the mitochondrial permeability …

H Uchino, R Minamikawa-Tachino, T Kristián… - Neurobiology of …, 2002 - Elsevier
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial …

H Uchino, R Minamikawa-Tachino… - Neurobiology of …, 2002 - scholar.archive.org
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

[引用][C] Differential neuroprotection by cyclosporin A and FK506 following ischemia corresponds with differing abilities to inhibit calcineurin and the mitochondrial …

H UCHINO - Neurobiol Dis, 2002 - cir.nii.ac.jp
Differential neuroprotection by cyclosporin A and FK506 following ischemia corresponds with
differing abilities to inhibit calcineurin and the mitochondrial permeability transition | CiNii …

Differential neuroprotection by cyclosporin A and FK506 following ischemia corresponds with differing abilities to inhibit calcineurin and the mitochondrial permeability …

H Uchino, R Minamikawa-Tachino… - Neurobiology of …, 2002 - pubmed.ncbi.nlm.nih.gov
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

[PDF][PDF] Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial …

H Uchino, R Minamikawa-Tachino, T Kristian… - Neurobiology of …, 2002 - academia.edu
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

Differential neuroprotection by cyclosporin A and FK506 following ischemia corresponds with differing abilities to inhibit calcineurin and the mitochondrial permeability …

H Uchino, R Minamikawa-Tachino, T Kristián… - Neurobiology of …, 2002 - europepmc.org
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial …

H Uchino, R Minamikawa-Tachino, T Kristián… - Neurobiology of …, 2002 - infona.pl
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

[引用][C] Differential neuroprotection by cyclosporine A and FK506 following ischemia corresponds with differing abilities to inhibit calcineurin and the mitochondrial …

H UCHINO - Neurobiol Dis, 2002 - cir.nii.ac.jp
Differential neuroprotection by cyclosporine A and FK506 following ischemia corresponds
with differing abilities to inhibit calcineurin and the mitochondrial permeability transition | CiNii …

[PDF][PDF] Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial …

H Uchino, R Minamikawa-Tachino, T Kristian… - Neurobiology of …, 2002 - academia.edu
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …

Differential Neuroprotection by Cyclosporin A and FK506 Following Ischemia Corresponds with Differing Abilities to Inhibit Calcineurin and the Mitochondrial …

H Uchino, R Minamikawa-Tachino, T Kristián… - Neurobiology of …, 2002 - infona.pl
Transient global or forebrain ischemia leads to severe brain damage following delayed
neuronal cell death. We previously reported that cyclosporin A (CsA) provides near total …