CHCHD10 mutations p.R15L and p.G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt, P Oeckl… - Human molecular …, 2018 - academic.oup.com
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency.
SJ Brockmann, A Freischmidt, P Oeckl… - Human Molecular …, 2018 - search.ebscohost.com
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
[引用][C] CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt, P Oeckl, K Mueller… - 2018 - oparu.uni-ulm.de
CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency.
SJ Brockmann, AM Helferich, C Paone… - Human molecular …, 2018 - pub.dzne.de
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt… - Human Molecular …, 2018 - epub.uni-regensburg.de
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
CHCHD10 mutations p.R15L and p.G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt, P Oeckl… - Human Molecular …, 2018 - academic.oup.com
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
[引用][C] CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt… - Human Molecular …, 2018 - researchportal.helsinki.fi
CHCHD10 mutations p.R15L and p.G66V cause motoneuron disease by haploinsufficiency —
University of Helsinki Skip to main navigation Skip to search Skip to main content University of …
University of Helsinki Skip to main navigation Skip to search Skip to main content University of …
CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt, P Oeckl… - Human Molecular …, 2018 - diva-portal.org
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
[引用][C] CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt, P Oeckl… - HUMAN …, 2018 - researchportal.tuni.fi
CHCHD10 mutations p.R15L and p.G66V cause motoneuron disease by haploinsufficiency
— Tampere University Research Portal Skip to main navigation Skip to search Skip to main …
— Tampere University Research Portal Skip to main navigation Skip to search Skip to main …
CHCHD10 mutations p. R15L and p. G66V cause motoneuron disease by haploinsufficiency
SJ Brockmann, A Freischmidt… - Human molecular …, 2018 - pubmed.ncbi.nlm.nih.gov
Mutations in the mitochondrially located protein CHCHD10 cause motoneuron disease by
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …
an unknown mechanism. In this study, we investigate the mutations p. R15L and p. G66V in …