[HTML][HTML] Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia, EC Tsilibary - Scientific reports, 2017 - nature.com
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease.
A Kaminari, N Giannakas, A Tzinia, EC Tsilibary - Scientific Reports, 2017 - europepmc.org
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia… - Scientific …, 2017 - ui.adsabs.harvard.edu
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
[引用][C] Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia, EC Tsilibary - Scientific Reports, 2017 - cir.nii.ac.jp
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in
the 5XFAD model of Alzheimer’s disease | CiNii Research CiNii 国立情報学研究所 学術情報 …
the 5XFAD model of Alzheimer’s disease | CiNii Research CiNii 国立情報学研究所 学術情報 …
[HTML][HTML] Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia… - Scientific Reports, 2017 - ncbi.nlm.nih.gov
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia… - Scientific reports, 2017 - experts.umn.edu
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease
A Kaminari, N Giannakas, A Tzinia… - Scientific …, 2017 - pubmed.ncbi.nlm.nih.gov
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
Overexpression of matrix metalloproteinase-9 (MMP-9) rescues insulin-mediated impairment in the 5XFAD model of Alzheimer's disease.
A Kaminari, N Giannakas, A Tzinia, EC Tsilibary - Scientific Reports, 2017 - europepmc.org
A hallmark of Alzheimer's disease (AD) is the accumulation of oligomeric amyloid-β (Aβ)
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …
peptide, which may be primarily responsible for neuronal dysfunction. Insulin signaling …