Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells

Q Li, Z Ye, J Wen, L Ma, Y He, G Lian, Z Wang… - Biochemical and …, 2009 - Elsevier
The tumor necrosis factor (TNF) can induce apoptosis in many cells including MCF-7 cells.
To identify the genes responsible for TNF-induced apoptosis, we generated a series of TNF …

Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells

Q Li, Z Ye, J Wen, L Ma, Y He, G Lian… - Biochemical and …, 2009 - pure.qub.ac.uk
The tumor necrosis factor (TNF) can induce apoptosis in many cells including MCF-7 cells.
To identify the genes responsible for TNF-induced apoptosis, we generated a series of TNF …

Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells

Q Li, Z Ye, J Wen, L Ma, Y He, G Lian, Z Wang… - Biochemical and …, 2009 - infona.pl
The tumor necrosis factor (TNF) can induce apoptosis in many cells including MCF-7 cells.
To identify the genes responsible for TNF-induced apoptosis, we generated a series of TNF …

Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells.

Q Li, Z Ye, J Wen, L Ma, Y He, G Lian… - Biochemical and …, 2009 - europepmc.org
The tumor necrosis factor (TNF) can induce apoptosis in many cells including MCF-7 cells.
To identify the genes responsible for TNF-induced apoptosis, we generated a series of TNF …

Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells

Q Li, Z Ye, J Wen, L Ma, Y He, G Lian… - Biochemical and …, 2009 - pubmed.ncbi.nlm.nih.gov
The tumor necrosis factor (TNF) can induce apoptosis in many cells including MCF-7 cells.
To identify the genes responsible for TNF-induced apoptosis, we generated a series of TNF …

[引用][C] Gelsolin, but not its cleavage, is required for TNF-induced ROS generation and apoptosis in MCF-7 cells

Q LI, Z YE, JUN WEN, LIN MA, Y HE, G LIAN… - Biochemical and …, 2009 - Elsevier