[HTML][HTML] Cellular prion protein as a receptor of toxic amyloid-β42 oligomers is important for Alzheimer's disease
Y Zhang, Y Zhao, L Zhang, W Yu, Y Wang… - Frontiers in cellular …, 2019 - frontiersin.org
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease.
Y Zhang, Y Zhao, L Zhang, W Yu… - Frontiers in Cellular …, 2019 - search.ebscohost.com
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
[引用][C] Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu, Y Wang… - Frontiers in Cellular …, 2019 - cir.nii.ac.jp
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for
Alzheimer’s Disease | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] …
Alzheimer’s Disease | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu… - Frontiers in cellular …, 2019 - pubmed.ncbi.nlm.nih.gov
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease.
Y Zhang, Y Zhao, L Zhang, W Yu, Y Wang… - Frontiers in Cellular …, 2019 - europepmc.org
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu… - … Editor's Pick 2021, 2021 - books.google.com
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
[HTML][HTML] Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu, Y Wang… - Frontiers in Cellular …, 2019 - ncbi.nlm.nih.gov
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu… - Frontiers in Cellular …, 2019 - search.proquest.com
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
Cellular Prion Protein as a Receptor of Toxic Amyloid-β42 Oligomers Is Important for Alzheimer's Disease
Y Zhang, Y Zhao, L Zhang, W Yu… - Frontiers in cellular …, 2019 - pubmed.ncbi.nlm.nih.gov
The pathological features of Alzheimer's disease (AD) include senile plaques induced by
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …
amyloid-β (Aβ) protein deposits, neurofibrillary tangles formed by aggregates of …