CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - Elsevier
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead mice (Crtap …
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[引用][C] CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N FRATZL-ZELMAN, R MORELLO, B LEE… - Bone (New York …, 2010 - pascal-francis.inist.fr
CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model
and in children with osteogenesis imperfecta type VII CNRS Inist Pascal-Francis CNRS …
[HTML] nih.gov

[HTML][HTML] CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with Osteogenesis Imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - ncbi.nlm.nih.gov
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead in mice …

CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - hero.epa.gov
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead mice (Crtap …

CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - infona.pl
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead mice (Crtap …
[PDF] amazonaws.com

[PDF][PDF] CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee… - …, 2010 - publicationslist.org.s3.amazonaws …
N. Fratzl-Zelman a,⁎, R. Morello b, 1, B. Lee b, c, F. Rauch d, FH Glorieux d, BM Misof a, K.
Klaushofer a, P. Roschger aa Ludwig Boltzmann Institute of Osteology at Hanusch Hospital …
[PDF] academia.edu

[PDF][PDF] CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with Osteogenesis Imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - academia.edu
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead in mice …

CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII.

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2009 - europepmc.org
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead in mice …

CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2010 - pubmed.ncbi.nlm.nih.gov
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead mice (Crtap …

CRTAP deficiency leads to abnormally high bone matrix mineralization in a murine model and in children with osteogenesis imperfecta type VII.

N Fratzl-Zelman, R Morello, B Lee, F Rauch… - Bone, 2009 - europepmc.org
Cartilage-associated protein (CRTAP) is an essential cofactor for the proper post-
translational chain modification and collagen folding. CRTAP mutations lead in mice …