ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the 4 kidney cortex of OVE26 mice 5
BM Ford, AA Eid, M Göőz, JL Barnes, YC Gorin… - 2013 - journals.physiology.org
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis 57 and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis 57 and decline in renal function. The pathogenic mechanisms of matrix …
[PDF][PDF] ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
BM Ford, AA Eid, M Göoz, JL Barnes… - Am J Physiol Renal …, 2013 - researchgate.net
MATERIALS AND METHODS Materials. TMI-005 was a gift from Pfizer-Wyeth
Pharmaceuticals. Tween-80, PEG-400, dimethyl sulfoxide, and D-glucose were purchased …
Pharmaceuticals. Tween-80, PEG-400, dimethyl sulfoxide, and D-glucose were purchased …
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice.
BM Ford, AA Eid, M Göőz, JL Barnes… - American Journal of …, 2013 - europepmc.org
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
BM Ford, AA Eid, M Göőz, JL Barnes… - American journal …, 2013 - pubmed.ncbi.nlm.nih.gov
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
[HTML][HTML] ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
BM Ford, AA Eid, M Göőz, JL Barnes… - American Journal of …, 2013 - ncbi.nlm.nih.gov
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
[引用][C] ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
BM FORD, AA EID, M GÖOZ… - American journal of …, 2013 - pascal-francis.inist.fr
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of
OVE26 mice CNRS Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases …
OVE26 mice CNRS Inist Pascal-Francis CNRS Pascal and Francis Bibliographic Databases …
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice
BM Ford, AA Eid, M Göőz, JL Barnes, YC Gorin… - American Journal of …, 2013 - cir.nii.ac.jp
抄録< jats: p> Matrix protein accumulation is a prominent feature of diabetic nephropathy
that contributes to renal fibrosis and decline in renal function. The pathogenic mechanisms …
that contributes to renal fibrosis and decline in renal function. The pathogenic mechanisms …
ADAM17 mediates Nox4 expression and NADPH oxidase activity in the kidney cortex of OVE26 mice.
BM Ford, AA Eid, M Göőz, JL Barnes… - American Journal of …, 2013 - europepmc.org
Matrix protein accumulation is a prominent feature of diabetic nephropathy that contributes
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …
to renal fibrosis and decline in renal function. The pathogenic mechanisms of matrix …