Calcium signaling, excitability, and synaptic plasticity defects in a mouse model of Alzheimer's disease

H Zhang, J Liu, S Sun, E Pchitskaya… - Journal of …, 2015 - content.iospress.com
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

[PDF][PDF] Calcium signaling, excitability and synaptic plasticity defects in mouse model of Alzheimer's disease

H Zhang, J Liu, S Sun, E Pchitskaya… - J Alzheimers …, 2015 - aictherapy.com
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

Calcium Signaling, Excitability, and Synaptic Plasticity Defects in a Mouse Model of Alzheimer's Disease

H Zhanga, J Liua, S Suna… - Journal of …, 2015 - content.iospress.com
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

Calcium signaling, excitability, and synaptic plasticity defects in a mouse model of Alzheimer's disease.

H Zhang, J Liu, S Sun, E Pchitskaya… - Journal of Alzheimer's …, 2015 - europepmc.org
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

[PDF][PDF] Calcium Signaling, Excitability, and Synaptic Plasticity Defects in a Mouse Model of Alzheimer's Disease

H Zhanga, J Liua, S Suna, E Pchitskayac… - Journal of Alzheimer's …, 2015 - medfiz.spbstu.ru
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

[PDF][PDF] Calcium Signaling, Excitability, and Synaptic Plasticity Defects in a Mouse Model of Alzheimer's Disease

H Zhanga, J Liua, S Suna, E Pchitskayac… - Journal of Alzheimer's …, 2015 - researchgate.net
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

[HTML][HTML] Calcium signaling, excitability and synaptic plasticity defects in mouse model of Alzheimer's disease

H Zhang, J Liu, S Sun, E Pchitskaya… - Journal of Alzheimer's …, 2015 - ncbi.nlm.nih.gov
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

Calcium signaling, excitability, and synaptic plasticity defects in a mouse model of Alzheimer's disease.

H Zhang, J Liu, S Sun, E Pchitskaya… - Journal of Alzheimer's …, 2015 - psycnet.apa.org
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

[PDF][PDF] Calcium signaling, excitability and synaptic plasticity defects in mouse model of Alzheimer's disease

H Zhang, J Liu, S Sun, E Pchitskaya, E Popugaeva… - J Alzheimers …, 2015 - academia.edu
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …

Calcium Signaling, Excitability, and Synaptic Plasticity Defects in a Mouse Model of Alzheimer's Disease

H Zhang, J Liu, S Sun… - Journal of …, 2015 - utsouthwestern.elsevierpure.com
Alzheimer's disease (AD) and aging result in impaired ability to store memories, but the
cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) …