Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection

MJ Scott, JJ Hoth, M Turina, DR Woods, WG Cheadle - Cytokine, 2006 - Elsevier
Interleukin (IL)-10 is an anti-inflammatory cytokine known to modulate the outcome of sepsis
by decreasing pro-inflammatory cytokine production, including IL-12, a main activator of …

Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection.

MJ Scott, JJ Hoth, M Turina, DR Woods, WG Cheadle - Cytokine, 2006 - europepmc.org
Interleukin (IL)-10 is an anti-inflammatory cytokine known to modulate the outcome of sepsis
by decreasing pro-inflammatory cytokine production, including IL-12, a main activator of …

Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection

MJ Scott, JJ Hoth, M Turina, DR Woods, WG Cheadle - Cytokine, 2006 - infona.pl
Interleukin (IL)-10 is an anti-inflammatory cytokine known to modulate the outcome of sepsis
by decreasing pro-inflammatory cytokine production, including IL-12, a main activator of …

[引用][C] Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection

M SCOTT, J HOTH, M TURINA, D WOODS… - Cytokine, 2006 - cir.nii.ac.jp
Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during
bacterial infection | CiNii Research CiNii 国立情報学研究所 学術情報ナビゲータ[サイニィ] 詳細へ …

Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection

MJ Scott, JJ Hoth, M Turina, DR Woods… - …, 2006 - pubmed.ncbi.nlm.nih.gov
Interleukin (IL)-10 is an anti-inflammatory cytokine known to modulate the outcome of sepsis
by decreasing pro-inflammatory cytokine production, including IL-12, a main activator of …

[引用][C] Interleukin-10 suppresses natural killer cell but not natural killer T cell activation during bacterial infection

MJ SCOTT, JJ HOTH, M TURINA, DR WOODS… - Cytokine, 2006 - Elsevier