miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic plasticity via AMPA and NMDA receptors

Y Xu, P Chen, X Wang, J Yao, S Zhuang - Neuroscience Letters, 2018 - Elsevier
Abstract MicroRNA (miR)-34a was recently determined to contribute to the pathological
development of Alzheimer's disease (AD). miR-34a deficiency significantly attenuates …
被引用次数：56 相关文章

[引用][C] miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic plasticity via AMPA and NMDA receptors

Y Xu, P Chen, X Wang, J Yao, S Zhuang - Neuroscience Letters, 2018 - cir.nii.ac.jp
miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic
plasticity via AMPA and NMDA receptors | CiNii Research CiNii 国立情報学研究所 学術情報 …

miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic plasticity via AMPA and NMDA receptors.

Y Xu, P Chen, X Wang, J Yao, S Zhuang - Neuroscience Letters, 2018 - europepmc.org
MicroRNA (miR)-34a was recently determined to contribute to the pathological development
of Alzheimer's disease (AD). miR-34a deficiency significantly attenuates cognitive deficits in …

miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic plasticity via AMPA and NMDA receptors

Y Xu, P Chen, X Wang, J Yao… - Neuroscience …, 2018 - pubmed.ncbi.nlm.nih.gov
MicroRNA (miR)-34a was recently determined to contribute to the pathological development
of Alzheimer's disease (AD). miR-34a deficiency significantly attenuates cognitive deficits in …