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Complement C3 deficiency leads to accelerated amyloid β plaque deposition and neurodegeneration and modulation of the microglia/macrophage phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook… - Journal of …, 2008 - Soc Neuroscience
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …
被引用次数：364 相关文章

[引用][C] Complement C3 Deficiency Leads to Accelerated Amyloid Plaque Deposition and Neurodegeneration and Modulation of the Microglia/Macrophage Phenotype …

M Maier, Y Peng, L Jiang, TJ Seabrook… - Journal of …, 2008 - cir.nii.ac.jp
Complement C3 Deficiency Leads to Accelerated Amyloid Plaque Deposition and
Neurodegeneration and Modulation of the Microglia/Macrophage Phenotype in Amyloid …
[PDF] academia.edu

[PDF][PDF] Complement C3-Deficiency Leads to Accelerated Aβ Plaque Deposition and Neurodegeneration, and Modulation of the Microglia/Macrophage Phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook, MC Carroll - J Neurosci, 2008 - academia.edu
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …

Complement C3 deficiency leads to accelerated amyloid beta plaque deposition and neurodegeneration and modulation of the microglia/macrophage phenotype in …

M Maier, Y Peng, L Jiang… - The Journal of …, 2008 - pubmed.ncbi.nlm.nih.gov
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …
[HTML] nih.gov

[HTML][HTML] Complement C3 Deficiency Leads to Accelerated Amyloid β Plaque Deposition and Neurodegeneration and Modulation of the Microglia/Macrophage …

M Maier, Y Peng, L Jiang, TJ Seabrook… - The Journal of …, 2008 - ncbi.nlm.nih.gov
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …
[PDF] researchgate.net

[PDF][PDF] Complement C3-Deficiency Leads to Accelerated Aβ Plaque Deposition and Neurodegeneration, and Modulation of the Microglia/Macrophage Phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook, MC Carroll - J Neurosci, 2008 - researchgate.net
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …

Complement C3 deficiency leads to accelerated amyloid beta plaque deposition and neurodegeneration and modulation of the microglia/macrophage phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook… - The Journal of …, 2008 - europepmc.org
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …
[PDF] jneurosci.org

[PDF][PDF] Complement C3 Deficiency Leads to Accelerated Amyloidß Plaque Deposition and Neurodegeneration and Modulation of the Microglia/Macrophage …

M Maier, Y Peng, L Jiang, TJ Seabrook… - The Journal of …, 2008 - Soc Neuroscience
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …

Complement C3 deficiency leads to accelerated amyloid beta plaque deposition and neurodegeneration and modulation of the microglia/macrophage phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook… - The Journal of …, 2008 - europepmc.org
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …
[HTML] nih.gov

[HTML][HTML] Complement C3-Deficiency Leads to Accelerated Aβ Plaque Deposition and Neurodegeneration, and Modulation of the Microglia/Macrophage Phenotype in …

M Maier, Y Peng, L Jiang, TJ Seabrook… - The Journal of …, 2008 - ncbi.nlm.nih.gov
Complement factor C3 is the central component of the complement system and a key
inflammatory protein activated in Alzheimer's disease (AD). Previous studies demonstrated …