Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, S Rayaprolu, EB Dammer… - Elife, 2022 - elifesciences.org
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

[HTML][HTML] Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, S Rayaprolu, EB Dammer… - eLife, 2022 - ncbi.nlm.nih.gov
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's.

VJ Olah, AM Goettemoeller, S Rayaprolu, EB Dammer… - Elife, 2022 - europepmc.org
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's.

VJ Olah, AM Goettemoeller, S Rayaprolu… - eLife, 2022 - search.ebscohost.com
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, R Sruti, EB Dammer… - eLife, 2022 - search.proquest.com
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, S Rayaprolu, EB Dammer… - 2022 - agris.fao.org
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv3 channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, S Rayaprolu… - …, 2022 - pubmed.ncbi.nlm.nih.gov
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

[PDF][PDF] Biophysical Kv channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Olah, AM Goettemoeller, S Rayaprolu, EB Dammer… - researchgate.net
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Oláh, AM Goettemoeller, J Dimidschstein… - bioRxiv, 2021 - biorxiv.org
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …

Biophysical Kv channel alterations dampen excitability of cortical PV interneurons and contribute to network hyperexcitability in early Alzheimer's

VJ Oláh, AM Goettemoeller, J Dimidschstein… - 2021 - europepmc.org
In Alzheimer's disease (AD), a multitude of genetic risk factors and early biomarkers are
known. Nevertheless, the causal factors responsible for initiating cognitive decline in AD …