The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism

M Heinrichs, DED Ashour, J Siegel… - Cardiovascular …, 2021 - academic.oup.com
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure—but …

The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism.

M Heinrichs, D Ashour, J Siegel, L Büchner… - Cardiovascular …, 2021 - europepmc.org
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure-but …

[PDF][PDF] The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism

M Heinrichs, DED Ashour, J Siegel, L Buchner… - researchgate.net
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure—but …

The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism

M Heinrichs, DED Ashour, J Siegel… - Cardiovascular …, 2021 - academic.oup.com
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure—but …

The healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism

M Heinrichs, DED Ashour, J Siegel… - Cardiovascular …, 2021 - pubmed.ncbi.nlm.nih.gov
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure-but …

healing myocardium mobilizes a distinct B-cell subset through a CXCL13-CXCR5-dependent mechanism.

M Heinrichs, DED Ashour, J Siegel… - Cardiovascular …, 2021 - search.ebscohost.com
Aims Recent studies have revealed that B cells and antibodies can influence inflammation
and remodelling following a myocardial infarction (MI) and culminating in heart failure—but …