Implication of Kir4. 1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4. 1 knock-out mice

O Chever, B Djukic, KD McCarthy… - Journal of …, 2010 - Soc Neuroscience
The Kir4. 1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

Implication of Kir4.1 Channel in Excess Potassium Clearance: AnIn VivoStudy on Anesthetized Glial-Conditional Kir4.1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - cir.nii.ac.jp
抄録< jats: p> The K< jats: sub> ir</jats: sub> 4.1 channel is crucial for the maintenance of
the resting membrane potential of glial cells, and it is believed to play a main role in the …

[PDF][PDF] Implication of Kir 4.1 Channel in Excess Potassium Clearance: AnIn VivoStudy on Anesthetized Glial-Conditional Kir 4.1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - Soc Neuroscience
The Kir4. 1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

Implication of Kir4. 1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4. 1 knock-out mice.

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - europepmc.org
The K ir 4.1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

[PDF][PDF] Implication of Kir 4.1 Channel in Excess Potassium Clearance: AnIn VivoStudy on Anesthetized Glial-Conditional Kir 4.1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - researchgate.net
The Kir4. 1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

[PDF][PDF] Implication of Kir 4.1 Channel in Excess Potassium Clearance: AnIn VivoStudy on Anesthetized Glial-Conditional Kir 4.1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - scholar.archive.org
The Kir4. 1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

Implication of Kir4. 1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4. 1 knock-out mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - pubmed.ncbi.nlm.nih.gov
The K (ir) 4.1 channel is crucial for the maintenance of the resting membrane potential of
glial cells, and it is believed to play a main role in the homeostasis of extracellular …

[PDF][PDF] Implication of Kir4. 1 Channel in Excess Potassium Clearance: An In Vivo Study on Anesthetized Glial-Conditional Kir4. 1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - cdr.lib.unc.edu
The Kir4. 1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

[HTML][HTML] Implication of Kir4. 1 Channel in Excess Potassium Clearance: An In Vivo Study on Anesthetized Glial-Conditional Kir4. 1 Knock-Out Mice

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - ncbi.nlm.nih.gov
The K ir 4.1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …

Implication of Kir4. 1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4. 1 knock-out mice.

O Chever, B Djukic, KD McCarthy… - The Journal of …, 2010 - europepmc.org
The K ir 4.1 channel is crucial for the maintenance of the resting membrane potential of glial
cells, and it is believed to play a main role in the homeostasis of extracellular potassium. To …